J
Johannes Stuttmann
Researcher at Martin Luther University of Halle-Wittenberg
Publications - 41
Citations - 1621
Johannes Stuttmann is an academic researcher from Martin Luther University of Halle-Wittenberg. The author has contributed to research in topics: Gene & Arabidopsis. The author has an hindex of 17, co-authored 34 publications receiving 1130 citations. Previous affiliations of Johannes Stuttmann include University of the Mediterranean & Max Planck Society.
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Journal ArticleDOI
Structural basis for signaling by exclusive EDS1 heteromeric complexes with SAG101 or PAD4 in plant innate immunity.
Stephan Wagner,Johannes Stuttmann,Steffen Rietz,Raphael Guerois,Raphael Guerois,Elena Brunstein,Jaqueline Bautor,Karsten Niefind,Jane E. Parker +8 more
TL;DR: Although there is evolutionary conservation of α/β hydrolase topology in all three proteins, a noncatalytic resistance mechanism is indicated and the respective N-terminal domains appear to facilitate binding of the essential EP domains to create novel interaction surfaces on the heterodimer.
Journal ArticleDOI
Interaction between SGT1 and Cytosolic/Nuclear HSC70 Chaperones Regulates Arabidopsis Immune Responses
Laurent D. Noël,Laurent D. Noël,Giuseppe Cagna,Johannes Stuttmann,Lennart Wirthmüller,Shigeyuki Betsuyaku,Claus-Peter Witte,Riyaz Bhat,Nathalie Pochon,Thomas Colby,Jane E. Parker +10 more
TL;DR: It is concluded that an H SC70-SGT1 chaperone complex is important for multiple plant environmental responses and that the evolutionarily conserved SGS domain of SGT1 is a key determinant of the HSC70–S GT1 association.
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A Coevolved EDS1-SAG101-NRG1 Module Mediates Cell Death Signaling by TIR-Domain Immune Receptors.
Dmitry Lapin,Viera Kovacova,Xinhua Sun,Joram A. Dongus,Deepak D. Bhandari,Patrick von Born,Jaqueline Bautor,Nina Guarneri,Jakub Rzemieniewski,Johannes Stuttmann,Andreas Beyer,Jane E. Parker +11 more
TL;DR: The data suggest that TNL-triggered cell death and pathogen growth restriction are determined by distinctive features of E DS1-SAG101 and EDS1-PAD4 complexes and that these signaling machineries coevolved with other components within plant species or clades to regulate downstream pathways in TNL immunity.
Journal ArticleDOI
Generation of chromosomal deletions in dicotyledonous plants employing a user-friendly genome editing toolkit.
Jana Ordon,Johannes Gantner,Jan Kemna,Lennart Schwalgun,Maik Reschke,Jana Streubel,Jens Boch,Johannes Stuttmann +7 more
TL;DR: Targeting of small deletions by paired nucleases represents a simple approach for the generation of mutant alleles segregating as size polymorphisms in subsequent generations of genome editing.
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Perturbation of Arabidopsis Amino Acid Metabolism Causes Incompatibility with the Adapted Biotrophic Pathogen Hyaloperonospora arabidopsidis
Johannes Stuttmann,Hans-Michael Hubberten,Steffen Rietz,Jagreet Kaur,Paul Muskett,Raphael Guerois,Paweł Bednarek,Rainer Hoefgen,Jane E. Parker +8 more
TL;DR: P perturbations in amino acid homeostasis render the mutant plants unsuitable as an infection substrate for Hpa, and data show that the plant host metabolic state can, in specific ways, influence the ability of adapted biotrophic strains to cause disease.