J
Jonas Anders
Researcher at Karolinska Institutet
Publications - 4
Citations - 697
Jonas Anders is an academic researcher from Karolinska Institutet. The author has contributed to research in topics: Glial cell line-derived neurotrophic factor & Proto-Oncogene Proteins c-ret. The author has an hindex of 4, co-authored 4 publications receiving 664 citations.
Papers
More filters
Journal ArticleDOI
Released GFRα1 potentiates downstream signaling, neuronal survival, and differentiation via a novel mechanism of recruitment of c-Ret to lipid rafts
Gustavo Paratcha,Fernanda Ledda,Louise Baars,Muriel Coulpier,Valerie Besset,Jonas Anders,Rizaldy P. Scott,Carlos F. Ibáñez +7 more
TL;DR: The results provide an explanation for the tissue distribution of GFRalpha1 and support the physiological importance of c-Ret activation in trans as a novel mechanism to potentiate and diversify the biological responses to GDNF.
Journal ArticleDOI
Molecular Modeling of the Extracellular Domain of the RET Receptor Tyrosine Kinase Reveals Multiple Cadherin-like Domains and a Calcium-binding Site
TL;DR: The models of the cadherin-like domains of human RET represent valuable tools with which to guide future site-directed mutagenesis studies aimed at identifying residues involved in ligand binding and receptor activation.
Journal ArticleDOI
Coordinated Activation of Autophosphorylation Sites in the RET Receptor Tyrosine Kinase IMPORTANCE OF TYROSINE 1062 FOR GDNF MEDIATED NEURONAL DIFFERENTIATION AND SURVIVAL
TL;DR: The catalytic and signaling activities of RET, a tyrosine kinase receptor for glial cell line-derived neurotrophic factor (GDNF), are controlled by the autophosphorylation of several tyrosines in the RET cytoplasmic domain this paper.
Coordinated Activation of Autophosphorylation Sites in the RET
TL;DR: The catalytic and signaling activities of RET, a tyrosine kinase receptor for glial cell line-derived neurotrophic factor (GDNF), are controlled by the autophosphorylation of several tyrosines in the RET cytoplasmic domain this article.