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Jong-Seok Kim

Researcher at Konyang University

Publications -  15
Citations -  177

Jong-Seok Kim is an academic researcher from Konyang University. The author has contributed to research in topics: Neuroprotection & Amyloid beta. The author has an hindex of 5, co-authored 15 publications receiving 64 citations.

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Red Ginseng Attenuates Aβ-Induced Mitochondrial Dysfunction and Aβ-mediated Pathology in an Animal Model of Alzheimer's Disease

TL;DR: In vitro and in vivo findings indicated that RGE significantly improves Aβ-induced mitochondrial pathology and significantly ameliorated AD-related pathology, such as Aβ deposition, gliosis, and neuronal loss, and deficits in adult hippocampal neurogenesis in brains with AD.
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Omega-3 Fatty Acid-Type Docosahexaenoic Acid Protects against Aβ-Mediated Mitochondrial Deficits and Pathomechanisms in Alzheimer's Disease-Related Animal Model.

TL;DR: In vitro and in vivo findings demonstrate that DHA attenuated AD-associated pathologies, such as mitochondrial impairment, Aβ accumulation, neuroinflammation, neuronal loss, and impairment of adult hippocampal neurogenesis.
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Therapeutic effects of non-saponin fraction with rich polysaccharide from Korean red ginseng on aging and Alzheimer's disease.

TL;DR: Wang et al. as mentioned in this paper investigated the effects of NFP from Korean red ginseng (KRG) on AD pathologies, such as Aβ deposition, neuroinflammation, neurodegeneration, mitochondrial dysfunction, and impaired adult hippocampal neurogenesis.
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mTOR-Dependent Role of Sestrin2 in Regulating Tumor Progression of Human Endometrial Cancer.

TL;DR: Upregulation of SESN2 statistically correlated with shorter overall survival and disease-free survival in patients with endometrial cancer, and a relationship between S ESN2, the mTORC1 pathway, and endometri cancer growth, suggesting SESn2 as a potential therapeutic target in endometrian cancer.
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Targeting TBK1 Attenuates LPS-Induced NLRP3 Inflammasome Activation by Regulating of mTORC1 Pathways in Trophoblasts.

TL;DR: In this paper, the effect of TBK1 inhibition on lipopolysaccharide (LPS)-induced NLRP3 inflammasome activation and its underlying mechanisms in human trophoblast cell lines and mouse placenta was investigated.