J
José Costa
Publications - 6
Citations - 216
José Costa is an academic researcher. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 6, co-authored 6 publications receiving 216 citations. Previous affiliations of José Costa include South University of Science and Technology of China & Southern University of Science and Technology.
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Global, regional and national burden of low back pain 1990–2019: A systematic analysis of the Global Burden of Disease study 2019
TL;DR: In this article , the authors comprehensively analyzed the global level and trends of prevalence, incidence and years lived with disability for low back pain (LBP) from 1990 to 2019 by age, sex and sociodemographic index (SDI).
Journal ArticleDOI
Global, regional and national burden of low back pain 1990–2019: A systematic analysis of the Global Burden of Disease study 2019
Zengwu Shao,Sheng Chen,Sheng Chen,Mingjue Chen,Huiling Cao,Huiling Cao,Xiaohao Wu,Sixiong Lin,Sixiong Lin,José Costa,José Costa,Chu Tao,Huiling Cao,Zengwu Shao,Guozhi Xiao +14 more
TL;DR: In this article, the authors comprehensively analyzed the global level and trends of prevalence, incidence and years lived with disability (YLDs) for low back pain (LBP) from 1990 to 2019 by age, sex and sociodemographic index (SDI).
Journal ArticleDOI
Kindlin-2 inhibits Nlrp3 inflammasome activation in nucleus pulposus to maintain homeostasis of the intervertebral disc
TL;DR: In this paper , the focal adhesion protein Kindlin-2 is shown to be highly expressed in the nucleus pulposus (NP), but not in the anulus fibrosus and the cartilaginous endplates, in the IVD tissues.
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Kindlin-2 deletion in osteoprogenitors causes severe chondrodysplasia and low-turnover osteopenia in mice
TL;DR: This study demonstrates an essential role of Kinlind-2 expression in osteoprogenitors in regulating skeletogenesis and bone mass accrual and homeostasis in mice and may define a novel therapeutic target for treatment of skeletal diseases, such as chondrodysplasia and osteoporosis.
Journal ArticleDOI
Kindlin-2 deletion in osteoprogenitors causes severe chondrodysplasia and low-turnover osteopenia in mice
José Costa,José Costa +1 more
TL;DR: Kindlin-2 was highly expressed in osteoprogenitors during endochondral ossification, and deletion of this protein impaired bone formation and bone mass accrual and homeostasis as discussed by the authors .