scispace - formally typeset
J

Josemir W. Sander

Researcher at UCL Institute of Neurology

Publications -  705
Citations -  44517

Josemir W. Sander is an academic researcher from UCL Institute of Neurology. The author has contributed to research in topics: Epilepsy & Population. The author has an hindex of 106, co-authored 680 publications receiving 39038 citations. Previous affiliations of Josemir W. Sander include University of Cincinnati & National Institute for Health Research.

Papers
More filters
Journal ArticleDOI

The excitatory amino acid antagonist d-CPP-ene (SDZ EAA-494) in patients with epilepsy

TL;DR: Serum concentrations of DCPP-ene were found to be unpredictable and higher than expected from pharmacokinetic data on normal subjects, and there was no clear relationship between serum concentrations and the severity of side-effects.
Journal ArticleDOI

Mortality in patients with epilepsy: a study of patients in long term residential care.

TL;DR: This study seems to confirm suggestions that mortality rates are higher in patients with epilepsy than in the general population, but prospective studies are warranted to ascertain underlying mechanisms.
Journal ArticleDOI

Cardiac arrhythmias during or after epileptic seizures

TL;DR: Seven distinct patterns of (post)ictal cardiac arrhythmias were identified: ictal asystole, bradycardia and AV-conduction block were self-limiting in all but one of the cases and seen during focal dyscognitive seizures.
Journal ArticleDOI

The use of antiepileptic drugs - Principles and practice

TL;DR: Up to 70% of people developing epilepsy may expect to become seizure free with optimum antiepileptic drug (AED) therapy, but the remaining 30% are the most difficult to treat.
Journal ArticleDOI

Multicentre search for genetic susceptibility loci in sporadic epilepsy syndrome and seizure types: a case-control study

TL;DR: Results suggest that many SNPs contribute to disease predisposition in an apparently population- specific manner, however, subtle differences in phenotyping across cohorts, combined with a poor understanding of how the underlying genetic component to epilepsy aligns with current phenotypic classifications, might also account for apparent population-specific genetic risk factors.