J
Joseph W. Starnes
Researcher at University of North Carolina at Greensboro
Publications - 61
Citations - 2762
Joseph W. Starnes is an academic researcher from University of North Carolina at Greensboro. The author has contributed to research in topics: Skeletal muscle & Endurance training. The author has an hindex of 27, co-authored 60 publications receiving 2613 citations. Previous affiliations of Joseph W. Starnes include University of Pennsylvania & University of Texas at Austin.
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Journal ArticleDOI
Rapamycin, But Not Resveratrol or Simvastatin, Extends Life Span of Genetically Heterogeneous Mice
Richard A. Miller,Richard A. Miller,David E. Harrison,Clinton M. Astle,Joseph A. Baur,Angela R. Boyd,Rafael de Cabo,Elizabeth Fernandez,Kevin Flurkey,Martin A. Javors,James F. Nelson,Carlos J. Orihuela,Scott D. Pletcher,Zelton D Sharp,David A. Sinclair,Joseph W. Starnes,J. Erby Wilkinson,Nancy L. Nadon,Randy Strong +18 more
TL;DR: Evaluation of rapamycin's effects on mice is likely to help delineate the role of the mammalian target ofRapamycin complexes in the regulation of aging rate and age-dependent diseases and may help to guide a search for drugs that retard some or all of the diseases of aging.
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Acute exercise can improve cardioprotection without increasing heat shock protein content.
TL;DR: It is concluded that acute bouts of exercise can produce cardioprotective effects without an elevation of HSP 72 and heat shock protein concentration.
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Exercise training improves cardiac function after ischemia in the isolated, working rat heart.
TL;DR: It is concluded that exercise training results in an intrinsic myocardial adaptation, allowing greater recovery of cardiac pump function after global ischemia in the isolated rat heart.
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Effects of body temperature during exercise training on myocardial adaptations
TL;DR: It is concluded that exercise training results in dynamic changes in cardioprotective proteins over time which are influenced by core temperature andCardioprotection resulting from chronic exercise appears to be due to increased HSP 70.
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Exercise training improves metabolic response after ischemia in isolated working rat heart.
TL;DR: Hearts from exercise-trained rats demonstrated an increased metabolic tolerance to ischemic-reperfusion damage, which may contribute to the improved postischemic functional recovery.