J
Josué M. Nava-Sedeño
Researcher at Dresden University of Technology
Publications - 12
Citations - 119
Josué M. Nava-Sedeño is an academic researcher from Dresden University of Technology. The author has contributed to research in topics: Cellular automaton & Random walk. The author has an hindex of 5, co-authored 11 publications receiving 83 citations. Previous affiliations of Josué M. Nava-Sedeño include BRICS.
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Hook length of the bacterial flagellum is optimized for maximal stability of the flagellar bundle.
Imke Spöring,Vincent A. Martinez,Christian Hotz,Jana Schwarz-Linek,Keara L. Grady,Josué M. Nava-Sedeño,Teun Vissers,Hanna M. Singer,Manfred Rohde,Carole Bourquin,Carole Bourquin,Haralampos Hatzikirou,Wilson C. K. Poon,Yann S. Dufour,Marc Erhardt,Marc Erhardt +15 more
TL;DR: The results suggest that the molecular ruler mechanism evolved to control flagellar hook growth to the optimal length consistent with efficient bundle formation, and the hook-length control mechanism is a prime example of how bacteria evolved elegant but robust mechanisms to maximize their fitness under specific environmental constraints.
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Cellular automaton models for time-correlated random walks: derivation and analysis.
TL;DR: The computational efficiency of cellular automata combined with the analytical results paves the way to explore the relevance of memory and anomalous diffusion for the dynamics of interacting cell populations, like confluent cell monolayers and cell clustering.
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Extracting cellular automaton rules from physical Langevin equation models for single and collective cell migration.
TL;DR: This paper introduces a method to obtain lattice-gas cellular automaton interaction rules from physically-motivated “off-lattice” Langevin equation models for migrating cells and derived the LGCA transition probability rule from the steady-state distribution of the off- lattice Fokker-Planck equation.
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Cellular automaton models for time-correlated random walks: derivation and analysis
TL;DR: In this article, a non-Markovian lattice-gas cellular automata model for moving agents with memory is proposed, where the reorientation probabilities are derived from velocity autocorrelation functions that are given a priori; in that respect their approach is ''data-driven''.
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Understanding fibrosis pathogenesis via modeling macrophage-fibroblast interplay in immune-metabolic context
Elisa Setten,Alessandra Castagna,Josué M. Nava-Sedeño,Jonathan Weber,Roberta Carriero,Andreas I. Reppas,Valery Volk,Jessica Schmitz,Wilfried Gwinner,Haralampos Hatzikirou,Friedrich Feuerhake,Massimo Locati +11 more
TL;DR: In this article , a comprehensive functional model that considers macrophage-fibroblasts interaction in the metabolic/immunologic context of fibrotic tissue has been set up.