J
Juana M. Pasquini
Researcher at University of Buenos Aires
Publications - 119
Citations - 3965
Juana M. Pasquini is an academic researcher from University of Buenos Aires. The author has contributed to research in topics: Myelin & Oligodendrocyte. The author has an hindex of 30, co-authored 113 publications receiving 3619 citations. Previous affiliations of Juana M. Pasquini include National Scientific and Technical Research Council.
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Journal ArticleDOI
The Effects of Prenatal Iron Deficiency and Risperidone Treatment on the Rat Frontal Cortex: A Proteomic Analysis
Lorna A. Farrelly,Maria Victoria Rosato-Siri,Melanie Föcking,Martín Gabriel Codagnone,Analía Reinés,Patrick Dicker,Kieran Wynne,Michael Farrell,Mary Cannon,Gerard Cagney,Juana M. Pasquini,David Cotter +11 more
TL;DR: Looking at the proteomic effects of pID in adulthood on the rat frontal cortex area (FCA) revealed changes in metabolic processes, including the tricyclic acid cycle, mitochondrial dysfunction, and P13K/Akt signaling.
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Incorporation of [3H]thymidine into DNA and of [35S]sulfate into sulfatides of oligodendroglial cells during development: effect of malnutrition.
TL;DR: The results suggest that starved rats seem to be unable to maintain normal synthesis of these galactolipids throughout the entire period of active myelinogenesis.
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Normal development of spinal axons in early embryo stages and posterior locomotor function is independent of GAL-1.
TL;DR: The results show that development of spinal axons as well as the locomotor abilities observed in adult mice are independent of Gal‐1, which further validates the use of lgals‐1‐/‐ mice to develop spinal cord‐ or traumatic brain injury models for the evaluation of the regenerative action ofGal‐1.
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Effect of thyroid dysfunction upon phospholipid composition and CDP-choline incorporation in mitochondria and microsomal fraction isolated from liver and brain of suckling rats.
TL;DR: The results indicate that both experimental conditions affect in a different way the structure and function of brain mitochondria and microsomal fractions, and give further support to the hypothesis that mitochondria have a certain degree of autonomy for the synthesis of phosphatidylcholine.
Journal Article
Regeneración axonal posterior a lesiones traumáticas de médula espinal. papel crítico de galectina-1*
TL;DR: In this article, the effects of Galectin-1 (Gal-1), an endogenous glycan-binding protein, abundantly present at inflammation and injury sites, were reviewed and shown that dimeric Gal-1 promotes functional recovery of spinal lesions by interfering with inhibitory signals triggered by Sema3A adhering to the NRP-1/PlexinA4 complex.