Julie Weidner

TL;DR: A new method is devised to identify both common and stress-specific mRNA subsets associated with P-bodies and it is shown that the Puf5p-dependent storage of a subset of mRNAs in P- bodies under glucose starvation may be beneficial with respect to chronological lifespan.

TL;DR: An overview of the current research on miRNAs in allergic diseases, including atopic dermatitis, allergic rhinitis, and asthma is given and how individual miRNas function in the regulation of immune responses in epithelial cells and specialized immune cells in response to different environmental factors and respiratory viruses is discussed.

TL;DR: This manuscript demonstrates for the first time that P-body (PB) formation in response to stress is a regulated process, and that at least two different pathways drive PB assembly.

TL;DR: An overview of the current research on miRNAs in allergic diseases, including atopic dermatitis, allergic rhinitis and asthma is given and the importance of research into miRNA function in allergy and asthma to improve knowledge of the molecular mechanisms involved in the pathogenesis of this heterogeneous group of diseases is highlighted.

TL;DR: It is shown that the polysome-associated mRNA-binding protein Scp160 interacts with P body components, such as the decapping protein Dcp2 and the scaffold protein Pat1, presumably, on polysomes, to prevent P body formation under normal growth conditions.