J
June Hariprashad
Researcher at Cornell University
Publications - 24
Citations - 1230
June Hariprashad is an academic researcher from Cornell University. The author has contributed to research in topics: Leishmania donovani & T cell. The author has an hindex of 16, co-authored 24 publications receiving 1190 citations. Previous affiliations of June Hariprashad include NewYork–Presbyterian Hospital.
Papers
More filters
Journal ArticleDOI
Fertilization and pregnancy outcome with intracytoplasmic sperm injection for azoospermic men
Gianpiero D. Palermo,Peter N. Schlegel,June Hariprashad,Berrin Ergün,Anna Mielnik,Nikica Zaninovic,Lucinda L. Veeck,Zev Rosenwaks +7 more
TL;DR: In cases of obstructive azoospermia, fertilization and pregnancy rates with epididymal spermatozoa were higher than those achieved using spermatozosa obtained from the testes of men with non-obstructive azonpermia.
Journal ArticleDOI
Chromosome analysis of epididymal and testicular sperm in azoospermic patients undergoing ICSI
TL;DR: Sperm of non-obstructive azoospermic men had a higher incidence of chromosomal abnormalities, of which sex chromosome aneuploidy was the most predominant, and genetic counselling should be offered to all couples considering infertility treatment by ICSI with testicular sperm.
Journal ArticleDOI
Interleukin 12 is effective treatment for an established systemic intracellular infection: experimental visceral leishmaniasis.
Henry W. Murray,June Hariprashad +1 more
TL;DR: Results extend the antimicrobial-inducing capacity of IL-12 beyond prophylaxis by indicating that IL- 12 can exert clear-cut therapeutic activity in an established intracellular infection.
Journal ArticleDOI
Incidence of sperm aneuploidy in relation to semen characteristics and assisted reproductive outcome.
TL;DR: The increased frequency of chromosomal abnormalities did not have a direct effect on the fertilization rate, pregnancy characteristics, or neonatal outcome in this study, and suboptimal semen samples had a higher incidence of aneuploidy.
Journal ArticleDOI
Effect of granulocyte-macrophage colony-stimulating factor in experimental visceral leishmaniasis.
TL;DR: A role for endogenous GM-CSF is identified in the initial host defense response to L. donovani, reemphasize the influxing monocyte as an effector cell, and indicate that GM- CSF can be used as an antileishmanial treatment.