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Jürgen Berger

Researcher at University of Hamburg

Publications -  61
Citations -  6461

Jürgen Berger is an academic researcher from University of Hamburg. The author has contributed to research in topics: Marfan syndrome & Cancer. The author has an hindex of 32, co-authored 60 publications receiving 6216 citations.

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Emergency Room Triage of Patients with Acute Chest Pain by Means of Rapid Testing for Cardiac Troponin T or Troponin I

TL;DR: Bedside tests for cardiac-specific troponins are highly sensitive for the early detection of myocardial-cell injury in acute coronary syndromes and allow rapid and safe discharge of patients with an episode of acute chest pain from the emergency room.
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The ACCESS Study: Evaluation of Acute Candesartan Cilexetil Therapy in Stroke Survivors

TL;DR: Early neurohumoral inhibition has similar beneficial effects in cerebral and in myocardial ischemia and when there is need for or no contraindication against early antihypertensive therapy, candesartan cilexetil is a safe therapeutic option according to the ACCESS results.
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Primary Prevention of Sudden Cardiac Death in Idiopathic Dilated Cardiomyopathy The Cardiomyopathy Trial (CAT)

TL;DR: In this paper, patients with idiopathic dilated cardiomyopathy and impaired left ventricular ejection fraction were randomly assigned to the implantation of an implantable cardioverter-defibrillator (ICD) or control.
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A randomized study of coronary angioplasty compared with bypass surgery in patients with symptomatic multivessel coronary disease. German Angioplasty Bypass Surgery Investigation (GABI)

TL;DR: In selected patients with multivessel coronary disease, PTCA and CABG as initial treatments resulted in equivalent improvement in angina after one year, however, in order to achieve similar clinical outcomes, the patients treated with P TCA were more likely to require further interventions and antianginal drugs, whereas the patients treating with CABGs were morelikely to sustain an acute myocardial infarction at the time of the procedure.
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Low catalase levels in the epidermis of patients with vitiligo.

TL;DR: A new hypothesis has been formulated for the pathogenesis of vitiligo based on a consistent reduction in levels of catalase compared to normal healthy controls of matched photo-skin types and a increase in the concentration of hydrogen peroxide in the epidermis of these patients.