K
Katerina K. Papachroni
Researcher at National and Kapodistrian University of Athens
Publications - 10
Citations - 762
Katerina K. Papachroni is an academic researcher from National and Kapodistrian University of Athens. The author has contributed to research in topics: Osteoblast & Cellular differentiation. The author has an hindex of 9, co-authored 10 publications receiving 694 citations. Previous affiliations of Katerina K. Papachroni include University of Edinburgh.
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Journal ArticleDOI
Mechanotransduction in osteoblast regulation and bone disease.
Katerina K. Papachroni,Demetrios N. Karatzas,Kostas A. Papavassiliou,Efthimia K. Basdra,Athanasios G. Papavassiliou +4 more
TL;DR: The molecular pathways and players activated by mechanical stimulation during osteoblastic growth, differentiation and activity in health are reviewed, and the role of mechanostimulatory approaches in treating various bone pathophysiologies is considered.
Journal ArticleDOI
Autoantibodies to alpha‐synuclein in inherited Parkinson’s disease
Katerina K. Papachroni,Natalia Ninkina,Angeliki Papapanagiotou,Georgios M. Hadjigeorgiou,Georgia Xiromerisiou,Alexandros Papadimitriou,Anastasios Kalofoutis,Vladimir L. Buchman +7 more
TL;DR: The presence of AAb against synuclein family members in the peripheral blood serum of Parkinson’s disease patients and control individuals is examined to hypothesise that these AAb could be involved in pathogenesis of the inherited form of PD.
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Signaling networks and transcription factors regulating mechanotransduction in bone.
Dionysios J. Papachristou,Katerina K. Papachroni,Efthimia K. Basdra,Athanasios G. Papavassiliou +3 more
TL;DR: The major signaling cascades and transcription factors that control bone and cartilage mechanobiology are considered and the influence of the mechanical microenvironment on the determination of skeletal morphology is discussed.
Journal ArticleDOI
Colon carcinogenesis: Learning from NF-κB and AP-1
TL;DR: In this perspective, inhibition of NF-kappaB and AP-1 signaling mechanisms has become a rational target in the development of novel therapeutic approaches against CRC.
Journal ArticleDOI
Neurons Expressing the Highest Levels of γ-Synuclein Are Unaffected by Targeted Inactivation of the Gene
Natalia Ninkina,Katerina K. Papachroni,Darren C. Robertson,Oliver Schmidt,Liz Delaney,Francis O'Neill,Felipe A. Court,Arnon Rosenthal,Susan M. Fleetwood-Walker,Alun M. Davies,Vladimir L. Buchman +10 more
TL;DR: The data suggest that like α- synuclein, γ-synuclein is dispensable for the development and function of the nervous system.