K
Kaya L. Andrews
Researcher at Washington University in St. Louis
Publications - 4
Citations - 368
Kaya L. Andrews is an academic researcher from Washington University in St. Louis. The author has contributed to research in topics: Alport syndrome & Glomerulonephritis. The author has an hindex of 4, co-authored 4 publications receiving 353 citations.
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Journal ArticleDOI
Transcriptional induction of slit diaphragm genes by Lmx1b is required in podocyte differentiation
Jeffrey H. Miner,Roy Morello,Kaya L. Andrews,Cong Li,Corinne Antignac,Andrey S. Shaw,Brendan Lee +6 more
TL;DR: The results indicate that reduced levels of proteins associated with foot processes and the glomerular slit diaphragm likely contribute, along with reduced Levels of GBM collagens, to the nephropathy associated with NPS.
Journal ArticleDOI
Quantitative trait loci influence renal disease progression in a mouse model of Alport syndrome.
TL;DR: It is shown that genetic background strongly influences the timing of onset of disease and rate of progression to ESRF in these mice, and the existence of modifier genes that influence disease progression is suggested.
Journal ArticleDOI
Gelatinase B (MMP-9) Is Not Essential in the Normal Kidney and Does Not Influence Progression of Renal Disease in a Mouse Model of Alport Syndrome
Kaya L. Andrews,Tomoko Betsuyaku,Sharon S. Rogers,J. Michael Shipley,Robert M. Senior,Jeffrey H. Miner +5 more
TL;DR: GelB does not have a discernible role in the normal kidney and gelB is not involved in the progression of glomerulonephritis in a mouse model of Alport syndrome, suggesting that gelB plays a role in kidney development in vivo.
Journal ArticleDOI
Cloning and Characterization of KPL2, a Novel Gene Induced during Ciliogenesis of Tracheal Epithelial Cells
Lawrence E. Ostrowski,Kaya L. Andrews,Pravin D. Potdar,Hironori Matsuura,Anton M. Jetten,Paul Nettesheim +5 more
TL;DR: KPL2 was undetectable in heart and liver samples, but was expressed in brain and testis, tissues that contain axonemal structures, suggesting that KPL2 plays an important role in the differentiation or function of ciliated cells in the airway.