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Keith N. Frayn
Researcher at University of Oxford
Publications - 291
Citations - 24844
Keith N. Frayn is an academic researcher from University of Oxford. The author has contributed to research in topics: Adipose tissue & Postprandial. The author has an hindex of 80, co-authored 290 publications receiving 23356 citations. Previous affiliations of Keith N. Frayn include Southampton General Hospital & French Institute of Health and Medical Research.
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Journal ArticleDOI
Calculation of substrate oxidation rates in vivo from gaseous exchange
TL;DR: It is shown that erroneous results are obtained in the presence of metabolic processes such as lipogenesis and gluconeogenesis, so that the apparently negative rates encountered in patients infused with glucose do quantitatively represent net rates of synthesis.
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Glycaemic index methodology.
Fred Brouns,Inger Björck,Keith N. Frayn,Alison L. Gibbs,Vincent Lang,G Slama,Thomas M.S. Wolever +6 more
TL;DR: The present review discusses the most relevant methodological considerations and highlights specific recommendations regarding number of subjects, sex, subject status, inclusion and exclusion criteria, pre-test conditions, CHO test dose, blood sampling procedures, sampling times, test randomisation and calculation of glycaemic response area under the curve.
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Fatty Acids, Obesity, and Insulin Resistance: Time for a Reevaluation
TL;DR: The relationship between systemic concentrations of NEFA and obesity/insulin resistance is examined and the vehicle by which triacylglycerol stored in adipose tissue is transported to its sites of utilization is recognized.
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Adipose tissue as a buffer for daily lipid flux
TL;DR: The phenotype, at least with regard to insulin resistance, is similar with both excess and deficiency of adipose tissue, which could provide a framework for understanding the action of the thiazolidinedione insulin-sensitizing agents.
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Gluteofemoral body fat as a determinant of metabolic health
TL;DR: Gluteofemoral fat's role as a determinant of health by the long-term entrapment of excess fatty acids, thus protecting from the adverse effects associated with ectopic fat deposition, is underlines.