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Keri Lestari

Researcher at Padjadjaran University

Publications -  44
Citations -  460

Keri Lestari is an academic researcher from Padjadjaran University. The author has contributed to research in topics: Medicine & Myristica fragrans. The author has an hindex of 9, co-authored 34 publications receiving 304 citations.

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Kaempferol-3-O-rhamnoside isolated from the leaves of Schima wallichii Korth. inhibits MCF-7 breast cancer cell proliferation through activation of the caspase cascade pathway

TL;DR: This study isolated kaempferol-3-O-rhamnoside as an active compound from the leaves of Schima wallichii Korth, a plant commonly consumed by non-human primates, and its anti-cancer activities, including its ability to induce apoptotic mechanisms, were investigated in MCF-7 breast cancer cells.
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Antibiotic resistance in sepsis patients: Evaluation and recommendation of antibiotic use

TL;DR: The high use of antibiotic with a high level resistance requires a policy to support its rational use and local microbial pattern based on site infection and pattern of antibiotics sensitivity test can be used as supporting data to optimize appropriateness of empirical antibiotics therapy in sepsis patients.
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Medication Adherence Contributes to an Improved Quality of Life in Type 2 Diabetes Mellitus Patients: A Cross-Sectional Study

TL;DR: Adherence to prescribed medication showed a positive effect on diabetes-specific QOL in patients and this result is important not only in developing intervention programs for patients but also in improving their QOL through sustainable health promotion.
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NAD- and NADPH-Contributing Enzymes as Therapeutic Targets in Cancer: An Overview.

TL;DR: The rationale for targeting the key enzymes that maintain the NAD/NADPH pool, and preclinical studies of targeting these enzymes in cancers, are provided.
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Induction of caspase cascade pathway by kaempferol-3-O-rhamnoside in LNCaP prostate cancer cell lines

TL;DR: The results showed that kaempferol-3-O-rhamnoside inhibits the proliferation of LNCaP cells in a dose-dependent manner by upregulating the expression of caspase-8, caspases-9, cospase-3 and poly (ADP-ribose) polymerase proteins.