K
Kishore B. Challagundla
Researcher at University of Nebraska Medical Center
Publications - 44
Citations - 1793
Kishore B. Challagundla is an academic researcher from University of Nebraska Medical Center. The author has contributed to research in topics: microRNA & Neuroblastoma. The author has an hindex of 16, co-authored 34 publications receiving 1117 citations. Previous affiliations of Kishore B. Challagundla include University of Nebraska–Lincoln & Oregon Health & Science University.
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Journal ArticleDOI
Exosome-Mediated Transfer of microRNAs Within the Tumor Microenvironment and Neuroblastoma Resistance to Chemotherapy
Kishore B. Challagundla,Petra Wise,Paolo Neviani,Haritha Chava,Mariam Murtadha,Tong Xu,Rebekah J. Kennedy,Cristina Ivan,Xinna Zhang,Ivan Vannini,Francesca Fanini,Dino Amadori,George A. Calin,Michael D. Hadjidaniel,Hiroyuki Shimada,Ambrose Jong,Robert C. Seeger,Shahab Asgharzadeh,Amir Goldkorn,Muller Fabbri +19 more
TL;DR: Data indicate a unique role of exosomic miR-21 andmiR-155 in the cross-talk between NBL cells and human monocytes in the resistance to chemotherapy, through a novel exosomal miR -21/TLR8-NF-кB/exosomicMiR- 155/TERF1 signaling pathway.
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Impact of Social Determinants of Health on the Emerging COVID-19 Pandemic in the United States
TL;DR: The social determinants of health (SDOH), and how they impact disadvantaged populations during times of crisis, will help governments to better manage health emergencies so that every individual has equal opportunity to staying healthy.
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Long non-coding RNAs are emerging targets of phytochemicals for cancer and other chronic diseases.
Shruti Mishra,Sumit Singh Verma,Vipin Rai,Nikee Awasthee,Srinivas Chava,Kam M. Hui,Alan Prem Kumar,Kishore B. Challagundla,Gautam Sethi,Subash C. Gupta +9 more
TL;DR: How the modulation of lncRNAs by phytochemicals produce therapeutic effects on cancer and other chronic diseases is discussed in this review.
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Positive regulation of p53 stability and activity by the deubiquitinating enzyme Otubain 1
TL;DR: It is shown that the ovarian tumour domain‐containing Ub aldehyde‐binding protein 1 (Otub1) is a novel p53 regulator that directly suppresses MDM2‐mediated p53 ubiquitination in cells and in vitro and reveals a novel function for Otub1 in regulating p53 stability and activity.
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Ribosomal Protein L11 Recruits miR-24/miRISC To Repress c-Myc Expression in Response to Ribosomal Stress
Kishore B. Challagundla,Xiao Xin Sun,Xiaoli Zhang,Tiffany DeVine,Qinghong Zhang,Rosalie C. Sears,Mu Shui Dai +6 more
TL;DR: The results identify a novel regulatory paradigm wherein L11 plays a critical role in controlling c-myc mRNA turnover via recruiting miRISC in response to ribosomal stress.