K
Kishore B.S. Pasumarthi
Researcher at Dalhousie University
Publications - 59
Citations - 10372
Kishore B.S. Pasumarthi is an academic researcher from Dalhousie University. The author has contributed to research in topics: Myocyte & Cell cycle. The author has an hindex of 25, co-authored 57 publications receiving 9775 citations. Previous affiliations of Kishore B.S. Pasumarthi include St. Boniface General Hospital & University of Manitoba.
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Journal ArticleDOI
Donor cell transplantation for myocardial disease: does it complement current pharmacological therapies?
TL;DR: The potential of various donor cell types in myocardial repair, mechanisms underlying functional improvement in cell-based therapies, as well as potential interactions between pharmacological and cell- based therapies are discussed.
Journal ArticleDOI
Cardiomyocyte Cell Cycle Activation Ameliorates Fibrosis in the Atrium
Hidehiro Nakajima,Hisako O. Nakajima,Klaus Dembowsky,Kishore B.S. Pasumarthi,Kishore B.S. Pasumarthi,Loren J. Field +5 more
TL;DR: The notion that cardiomyocyte cell cycle induction can antagonize fibrosis in the myocardium is supported, as it is shown that atrial fibrosis was accompanied with carduomyocyte apoptosis.
Journal Article
Signaling mechanisms regulating fibroblast activation, phenoconversion and fibrosis in the heart.
TL;DR: A brief review of signaling mechanisms responsible for phenoconversion of CFs to CMFs is provided and critical targets for the treatment of cardiac fibrosis are identified.
Journal ArticleDOI
Quantification of cardiac fibrosis by colour-subtractive computer-assisted image analysis.
TL;DR: The results suggest that CD1 mice are an ideal model system to study catecholamine‐induced cardiac remodelling, as well as to screen candidate antifibrotic agents for future therapies.
Journal ArticleDOI
Ultrastructural and immunocharacterization of undifferentiated myocardial cells in the developing mouse heart.
TL;DR: It is suggested that embryonic myocardial cell differentiation is a gradual process and undifferentiated cells expressing Nkx2.5 in post‐chamber myocardium may represent a progenitor cell population while cells expressingNkx 2.5 and ANF represent differentiating myocytes.