L
Lars Karlsson
Researcher at Linköping University
Publications - 184
Citations - 5493
Lars Karlsson is an academic researcher from Linköping University. The author has contributed to research in topics: Mobile robot & Robot. The author has an hindex of 37, co-authored 174 publications receiving 4841 citations. Previous affiliations of Lars Karlsson include University of Gothenburg & Örebro University.
Papers
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A review of unsupervised feature learning and deep learning for time-series modeling ☆
TL;DR: This paper overviews the particular challenges present in time-series data and provides a review of the works that have either applied time- series data to unsupervised feature learning algorithms or alternatively have contributed to modifications of featurelearning algorithms to take into account the challenges present.
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A Potent and Selective Histamine H4 Receptor Antagonist with Anti-Inflammatory Properties
Robin L. Thurmond,Pragnya J. Desai,Paul J. Dunford,Wai-Ping Fung-Leung,Claudia L. Hofstra,Wen Jiang,Steven Nguyen,Jason P. Riley,Siquan Sun,Kacy N. Williams,James P. Edwards,Lars Karlsson +11 more
TL;DR: The results indicate that the histamine H4 receptor plays a role in the inflammatory process and may have the potential to be useful in treating inflammation in humans.
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Sleep stage classification using unsupervised feature learning
TL;DR: The use of an unsupervised feature learning architecture called deep belief nets (DBNs) is proposed and how to apply it to sleep data in order to eliminate the use of handmade features is shown.
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Nonclassical MHC Class II Molecules
TL;DR: Major histocompatibility complex (MHC) class II molecules are cell surface proteins that present peptides to CD4(+) T cells and the association with DM is essential for the intracellular transport of DO, and the two molecules remain associated in the endosomal system.
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KIR expression on self-reactive CD8 + T cells is controlled by T-cell receptor engagement
Bertrand Huard,Lars Karlsson +1 more
TL;DR: The data indicate that KIR expression on CD8+ T cells in vivo may be maintained through continuous encounters with antigen, as KIR-mediated inhibition of T-cell activation can be bypassed at high antigen concentrations, thus enabling them to mediate potentially useful immune functions to quantitatively or qualitatively different antigens.