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Laura Calosi

Researcher at University of Florence

Publications -  27
Citations -  314

Laura Calosi is an academic researcher from University of Florence. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 8, co-authored 21 publications receiving 221 citations.

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Therapeutic effects of the superoxide dismutase mimetic compound MnIIMe2DO2A on experimental articular pain in rats.

TL;DR: MnL4 appears as a novel protective compound potentially suitable for the treatment of joint diseases by behaving as a potent pain reliever in acute inflammatory and chronic articular pain.
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HYDAMTIQ, a selective PARP-1 inhibitor, improves bleomycin-induced lung fibrosis by dampening the TGF-β/SMAD signalling pathway.

TL;DR: The proposal that PARP inhibitors could have a therapeutic potential in reducing the progression of signs and symptoms of the disease by decreasing T GF‐β expression and the TGF‐β/SMAD transduction pathway is supported.
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Glucagon-like peptide 2 counteracts the mucosal damage and the neuropathy induced by chronic treatment with cisplatin in the mouse gastric fundus.

TL;DR: Glucagon‐like peptide‐2 (GLP‐2) is a pleiotropic hormone synthesized and secreted by the enteroendocrine ‘L’ cells able to exert intestine‐trophic and anti‐inflammatory effects.
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Protection from cigarette smoke-induced vascular injury by recombinant human relaxin-2 (serelaxin).

TL;DR: It is demonstrated that RLX is capable of counteracting CS‐mediated vascular damage and dysfunction by reducing oxidative stress, thus adding a tile to the growing mosaic of the beneficial effects of RLX in CVD.
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Immunohistochemical analysis of dendritic cells in skin lesions: Correlations with survival time

TL;DR: In this paper, the early response of Dendritic cells to skin wounding has been investigated in humans, and the results suggest that skin dendritic cell including Langerhans cells, including mast cells, participate to early response to wounding in concert with mast cells and that subpapillary blood vessels are primary sites of cell infiltration during that response in humans.