L
Luc Rochette
Researcher at University of Burgundy
Publications - 349
Citations - 10029
Luc Rochette is an academic researcher from University of Burgundy. The author has contributed to research in topics: Oxidative stress & Ischemia. The author has an hindex of 49, co-authored 326 publications receiving 8672 citations. Previous affiliations of Luc Rochette include French Institute of Health and Medical Research & University of Strathclyde.
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Dissociation between vascular oxidative stress and cardiovascular function in Wistar Kyoto and spontaneously hypertensive rats.
Pierre Sicard,Alexandra Oudot,Jean-Claude Guilland,Daniel Moreau,Catherine Vergely,Luc Rochette +5 more
TL;DR: Treatment with high doses of vitamin C in SHR can limit over-production of reactive oxygen species; however this effect was not accompanied with changes in arterial pressure and protection against I-R dysfunctions, which may suggest that association between vascular oxidative stress and cardiovascular function may be evoked.
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Non-genomic actions of thyroid hormones: Molecular aspects
Journal Article
Modification of vitamin E during ischemia-reperfusion in rat retina.
TL;DR: Vitamin E decreased only in the reperfused groups, demonstrating a depletion of defenses against free radical production occurring during reperfusion and a potential parameter to evaluate drugs in vitro.
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Effects of alterations in sympathetic nervous activity on the severity of reperfusion-induced arrhythmias in anaesthetised rats.
TL;DR: The results do not suggest an important role for sympathetic nervous activity in the genesis of reperfusion-induced arrhythmias in anaesthetised rats.
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Assessment of the cytoprotective role of adenosine in an in vitro cellular model of myocardial ischemia.
Sandrine Bès,Blandine Ponsard,Mounia El Asri,Cindy Tissier,David Vandroux,Luc Rochette,Pierre Athias +6 more
TL;DR: It appeared that cardiomyocytes possess both functional adenosine A(1) and A(2) receptors and that only the activation of adenosin A( 1) receptor had a cytoprotective effect against simulated ischemia-induced cardiac cell injury.