L
Luciano Mignini
Researcher at University of Birmingham
Publications - 31
Citations - 2894
Luciano Mignini is an academic researcher from University of Birmingham. The author has contributed to research in topics: Systematic review & Medicine. The author has an hindex of 14, co-authored 26 publications receiving 2583 citations.
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Reference EntryDOI
Episiotomy for vaginal birth
TL;DR: Restrictive episiotomy policies appear to have a number of benefits compared to routine episiotome policies, and there was an increased risk of anterior perineal trauma with restrictive episiotomies.
Journal ArticleDOI
Comparison of treatment effects between animal experiments and clinical trials: systematic review
Pablo Perel,Ian Roberts,Emily S. Sena,Philipa Wheble,Catherine Briscoe,Peter Sandercock,Malcolm R. Macleod,Luciano Mignini,Pradeep Jayaram,Khalid S. Khan +9 more
TL;DR: Conordance between treatment effects in animal experiments and clinical trials may be due to bias or to the failure of animal models to mimic clinical disease adequately.
Journal ArticleDOI
Factors predisposing women to chronic pelvic pain: systematic review
TL;DR: Several gynaecological and psychosocial factors are strongly associated with chronic pelvic pain and Randomised controlled trials of interventions targeting these potentially modifiable factors are needed to assess their clinical relevance in Chronic pelvic pain.
Journal ArticleDOI
Epilepsy in pregnancy and reproductive outcomes: a systematic review and meta-analysis
Luz Viale,John Allotey,Fiona Cheong-See,David Arroyo-Manzano,Dougall McCorry,Manny Bagary,Luciano Mignini,Khalid S. Khan,Javier Zamora,Shakila Thangaratinam +9 more
TL;DR: A small but significant association of epilepsy, exposure to antiepileptic drugs, and adverse outcomes exists in pregnancy, and this increased risk should be taken into account when counselling women with epilepsy.
Journal ArticleDOI
Mapping the theories of preeclampsia: the role of homocysteine.
TL;DR: Homocysteine concentrations are slightly increased in normotensive pregnancies that later develop preeclampsia and are considerably increased once preeclampia is established, but the observed association cannot be considered causal from the current literature.