M
M. Asif Amin
Researcher at University of Michigan
Publications - 40
Citations - 1823
M. Asif Amin is an academic researcher from University of Michigan. The author has contributed to research in topics: Arthritis & Angiogenesis. The author has an hindex of 21, co-authored 38 publications receiving 1592 citations. Previous affiliations of M. Asif Amin include Northwestern University.
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Journal ArticleDOI
Fractalkine: a novel angiogenic chemokine in rheumatoid arthritis.
Michael V. Volin,Michael V. Volin,James M. Woods,M. Asif Amin,Matthew A. Connors,Lisa A. Harlow,Alisa E. Koch +6 more
TL;DR: A new function is established for fkn as an angiogenic mediator and it is suggested that it may mediate angiogenesis in RA.
Journal ArticleDOI
Migration Inhibitory Factor Mediates Angiogenesis via Mitogen-Activated Protein Kinase and Phosphatidylinositol Kinase
M. Asif Amin,Olga V. Volpert,James M. Woods,Pawan Kumar,Lisa A. Harlow,Alisa E. Koch,Alisa E. Koch +6 more
TL;DR: It is demonstrated that in vitro rhMIF induces migration and tube formation in Matrigel of human dermal microvascular endothelial cells (HMVECs), with potency comparable to that of basic fibroblast growth factor.
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Migration inhibitory factor up-regulates vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 via Src, PI3 kinase, and NFκB
M. Asif Amin,Christian S. Haas,Kui Zhu,Pamela J. Mansfield,Michael J. Kim,Nicholas P. Lackowski,Alisa E. Koch +6 more
TL;DR: MIF and MIF-dependent signaling pathways may be a potential target for treating diseases characterized by up-regulation of cell adhesion molecules.
Journal ArticleDOI
Amelioration of rat adjuvant-induced arthritis by Met-RANTES.
Shiva Shahrara,Amanda E. I. Proudfoot,James M. Woods,Jeffrey H. Ruth,M. Asif Amin,Christy C. Park,Christian S. Haas,Richard M. Pope,G. Kenneth Haines,Yuan Yuan Zha,Alisa E. Koch +10 more
TL;DR: The data suggest that the targeting of Th1-associated chemokine receptors reduce joint inflammation, bone destruction, and cell recruitment into joints with AIA.
Journal ArticleDOI
CXCL16-mediated cell recruitment to rheumatoid arthritis synovial tissue and murine lymph nodes is dependent upon the MAPK pathway.
Jeffrey H. Ruth,Christian S. Haas,Christy C. Park,M. Asif Amin,Rita J. Martinez,G. Kenneth Haines,Shiva Shahrara,Phillip L. Campbell,Alisa E. Koch,Alisa E. Koch +9 more
TL;DR: Results point to a unique role for CXCL16 as a premier MNC recruiter in RA and suggest additional therapeutic possibilities, targeting CxCL16, its receptor, or its signaling pathways.