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Marcelo Pedrosa Gomes

Researcher at Federal University of Paraná

Publications -  90
Citations -  2219

Marcelo Pedrosa Gomes is an academic researcher from Federal University of Paraná. The author has contributed to research in topics: Biology & Glyphosate. The author has an hindex of 20, co-authored 69 publications receiving 1429 citations. Previous affiliations of Marcelo Pedrosa Gomes include Universidade Federal de Minas Gerais & Université du Québec à Montréal.

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Alteration of plant physiology by glyphosate and its by-product aminomethylphosphonic acid: an overview

TL;DR: The present review provides an overall picture of alterations of plant physiology caused by environmental exposure to glyphosate and its metabolite AMPA, and summarizes their effects on several physiological processes, particularly on photosynthesis, from photochemical events to C assimilation and translocation, as well as oxidative stress.
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Ecophysiological and anatomical changes due to uptake and accumulation of heavy metal in Brachiaria decumbens

TL;DR: Evaluating the effect of levels of soil contamination on biomass production, on photosynthetic characteristics and on anatomical changes in roots and leaves of brachiaria indicates that B. decumbens seems to have some degree of heavy metal tolerance.
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Differential effects of glyphosate and aminomethylphosphonic acid (AMPA) on photosynthesis and chlorophyll metabolism in willow plants.

TL;DR: It is observed that AMPA and glyphosate trigger different mechanisms leading to decreases in chlorophyll content and photosynthesis rates in willow plants, which may be due to various glyphosate:AMPA ratios in those plants.
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Reactive oxygen species and seed germination

TL;DR: Reactive oxygen species (ROS) are continuously produced by the metabolically active cells of seeds, and apparently play important roles in biological processes such as germination and dormancy.
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Ciprofloxacin induces oxidative stress in duckweed (Lemna minor L.): Implications for energy metabolism and antibiotic-uptake ability.

TL;DR: The results indicate that instead of being a photosystem II binding molecule, Cipro induces oxidative stress by targeting the mitochondrial ETC, which would explain the observed effects of the antibiotic on non-target eukaryotic organisms.