M
Maria Dolores Sanchez-Niño
Researcher at Autonomous University of Madrid
Publications - 179
Citations - 36178
Maria Dolores Sanchez-Niño is an academic researcher from Autonomous University of Madrid. The author has contributed to research in topics: Kidney disease & Kidney. The author has an hindex of 50, co-authored 164 publications receiving 27206 citations. Previous affiliations of Maria Dolores Sanchez-Niño include Carlos III Health Institute & Hospital Universitario La Paz.
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Journal ArticleDOI
The Role of PGC-1α and Mitochondrial Biogenesis in Kidney Diseases.
Miguel Fontecha-Barriuso,Diego Martin-Sanchez,Julio M. Martinez-Moreno,María Monsalve,Adrian M. Ramos,Maria Dolores Sanchez-Niño,Marta Ruiz-Ortega,Alberto Ortiz,Ana Belen Sanz +8 more
TL;DR: Low P GC-1α activity appears to be a common feature of AKI and CKD and recent characterization of nephroprotective approaches that increase PGC-1 α activity may pave the way for nephROprotective strategies potentially effective in both AKIand CKD.
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BASP1 Promotes Apoptosis in Diabetic Nephropathy
Maria Dolores Sanchez-Niño,Ana Belen Sanz,Corina Lorz,Andrea Gnirke,Maria Pia Rastaldi,Viji Nair,Jesús Egido,Marta Ruiz-Ortega,Matthias Kretzler,Alberto Ortiz +9 more
TL;DR: A combined functional genomics approach identified BASP1 as a proapoptotic factor in DN and possibly also in hypertensive nephropathy.
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Tweak induces proliferation in renal tubular epithelium: a role in uninephrectomy induced renal hyperplasia
Ana Belen Sanz,Maria Dolores Sanchez-Niño,María Concepción Izquierdo,Aniela Jakubowski,Pilar Justo,Luis Miguel Blanco-Colio,Marta Ruiz-Ortega,Jesús Egido,Alberto Ortiz +8 more
TL;DR: In this regard, TWEAK knock‐out mice with AKI displayed less tubular apoptosis and proliferation, as well as improved renal function, which may be relevant for compensatory renal hyperplasia following nephrectomy.
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Mitochondria-targeted therapies for acute kidney injury.
Luis Carlos Tábara,Jonay Poveda,Catalina Martin-Cleary,Rafael Selgas,Alberto Ortiz,Maria Dolores Sanchez-Niño +5 more
TL;DR: The morphological and functional mitochondrial changes during AKI are reviewed, as well as changes in the expression of mitochondrial genes and proteins, and the current status of novel therapeutic strategies specifically targeting mitochondria are summarized.
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Targeting epigenetic DNA and histone modifications to treat kidney disease.
Miguel Fontecha-Barriuso,Diego Martin-Sanchez,Olga Ruiz-Andres,Jonay Poveda,Maria Dolores Sanchez-Niño,Lara Valiño-Rivas,Marta Ruiz-Ortega,Alberto Ortiz,Ana Belen Sanz +8 more
TL;DR: While research into epigenetic modulators may provide novel therapies for kidney disease, caution should be exercised based on the clinical nephrotoxicity of some drugs.