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Maria Dolores Sanchez-Niño

Researcher at Autonomous University of Madrid

Publications -  179
Citations -  36178

Maria Dolores Sanchez-Niño is an academic researcher from Autonomous University of Madrid. The author has contributed to research in topics: Kidney disease & Kidney. The author has an hindex of 50, co-authored 164 publications receiving 27206 citations. Previous affiliations of Maria Dolores Sanchez-Niño include Carlos III Health Institute & Hospital Universitario La Paz.

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Global, regional, and national levels of maternal mortality, 1990–2015: a systematic analysis for the Global Burden of Disease Study 2015

Nicholas J Kassebaum, +539 more
TL;DR: In this article, the authors quantify maternal mortality throughout the world by underlying cause and age from 1990 to 2015, and assess the progress toward reducing maternal mortality to identify areas of success, remaining challenges, and frame policy discussions.
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The Inflammatory Cytokines TWEAK and TNFα Reduce Renal Klotho Expression through NFκB

Juan Antonio Moreno, +12 more
- 01 Jul 2011 - 
TL;DR: Inflammatory cytokines, such as TWEAK and TNFα, downregulate Klotho expression through an NFκB-dependent mechanism, which may partially explain the relationship between inflammation and diseases characterized by accelerated aging of organs, including CKD.
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Ferroptosis, but Not Necroptosis, Is Important in Nephrotoxic Folic Acid–Induced AKI

Diego Martin-Sanchez, +10 more
- 01 Jan 2017 - 
TL;DR: It is shown that ferrostatin-1 (Fer-1), an inhibitor of ferroptosis, preserved renal function and decreased histologic injury, oxidative stress, and tubular cell death in this model, and that immunogenicity secondary to ferroPTosis may further worsen the damage, although necroptosis-related proteins may have additional roles in AKI.
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Tenofovir Nephrotoxicity: 2011 Update

Beatriz Fernandez-Fernandez, +12 more
- 07 Jun 2011 - 
TL;DR: Despite initial cell culture and clinical trials results supporting the renal safety of tenofovir, its clinical use is associated with a low, albeit significant, risk of kidney injury, and regular monitoring of proximal tubular dysfunction and serum creatinine in high-risk patients is required to minimize nephrotoxicity.
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Globotriaosylsphingosine actions on human glomerular podocytes: implications for Fabry nephropathy

Maria Dolores Sanchez-Niño, +8 more
- 01 Jun 2011 - 
TL;DR: Lyso-Gb3 may have a role in glomersular injury in Fabry disease by promoting the release of secondary mediators of glomerular injury common to diabetic nephropathy, raising the issue of vitamin D receptor activation as potential adjunctive therapy in Fabries disease.