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Marie-Christine Mariol

Researcher at University of Lyon

Publications -  22
Citations -  951

Marie-Christine Mariol is an academic researcher from University of Lyon. The author has contributed to research in topics: Duchenne muscular dystrophy & Dystrophin. The author has an hindex of 17, co-authored 21 publications receiving 884 citations. Previous affiliations of Marie-Christine Mariol include French Institute of Health and Medical Research & Centre national de la recherche scientifique.

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DWnt4 regulates cell movement and focal adhesion kinase during Drosophila ovarian morphogenesis.

TL;DR: It is demonstrated that Drosphila Wnt4 is required for cell movement and FAK regulation during ovarian morphogenesis and the data suggest that DWnt4 facilitates motility through regulation of focal adhesions.
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The L-type voltage-dependent Ca2+ channel EGL-19 controls body wall muscle function in Caenorhabditis elegans.

TL;DR: It is demonstrated that body wall muscle cells generate spontaneous spike potentials and develop graded action potentials in response to injection of positive current of increasing amplitude and given evidence that the Ca2+ channel involved belongs to the L-type class and corresponds to EGL-19, a putative Ca1+ channel originally thought to be a member of this class on the basis of genomic data.
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DWnt-4, a novel Drosophila Wnt gene acts downstream of homeotic complex genes in the visceral mesoderm.

TL;DR: A novel Drosophila Wnt gene is isolated, DWnt-4, which maps close to wingless, suggesting that the two Wnt genes derive from a duplication that occurred early in evolution, since they are significantly diverged in sequence and structure.
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Mobilization of the gypsy and copia retrotransposons in Drosophila melanogaster induces reversion of the ovoD dominant female-sterile mutations: molecular analysis of revertant alleles

TL;DR: The ovo locus is required for the maintenance of the female germ line in Drosophila melanogaster and it is found that this strain contains an inordinately high number of gypsy transposable elements, and crossing it with the ovoD strains results in the mobilization of both gypsy and copia, with high‐frequency insertions into the oVO locus.
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Muscular degeneration in the absence of dystrophin is a calcium-dependent process.

TL;DR: It is shown that, in Caenorhabditis elegans, a gain-of-function mutation in the egl-19 calcium channel gene dramatically increases muscle degeneration in dystrophin mutants, and RNAi-mediated inhibition of eGL-19 function reduces Muscle degeneration by half.