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Mark S. Pearce

Researcher at Newcastle University

Publications -  306
Citations -  15432

Mark S. Pearce is an academic researcher from Newcastle University. The author has contributed to research in topics: Population & Birth weight. The author has an hindex of 56, co-authored 293 publications receiving 13537 citations. Previous affiliations of Mark S. Pearce include University of Newcastle & National Institute for Health Research.

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Survival adjusted cancer risks attributable to radiation exposure from cardiac catheterisations in children

TL;DR: Based on recent UK radiation dose levels, the risk of cancer following cardiac catheterisations is relatively low and strongly modified by survival and the type of procedure.
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Paternal occupational exposure to electro-magnetic fields as a risk factor for cancer in children and young adults: a case-control study from the North of England.

TL;DR: This work investigated whether an association exists between paternal occupations at birth involving such exposures and cancer risk in offspring, using data from the Northern Region Young Persons' Malignant Disease Registry (NRYPMDR).
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Reduction in radiation doses from paediatric CT scans in Great Britain

TL;DR: It is found that mAs for head and trunk CTs was approximately halved starting around 1990, and age-specific mAs was generally used for paediatric scans after this date, which will have substantially reduced the radiation exposure to children from CT scans in Great Britain.
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The impact of socio-economic status and mobility on perceived family functioning

TL;DR: In this paper, the authors investigated perceptions of current family functioning in relation to current household income level, educational status, social-class at birth and social mobility over the lifecourse.
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Paternal Occupational Exposure to Pesticides or Herbicides as Risk Factors for Cancer in Children and Young Adults: A Case-Control Study From the North of England

TL;DR: Results do not support a role for preconception paternal occupational exposures to pesticides or herbicides in the etiology of childhood cancer.