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Mark S. Pearce

Researcher at Newcastle University

Publications -  306
Citations -  15432

Mark S. Pearce is an academic researcher from Newcastle University. The author has contributed to research in topics: Population & Birth weight. The author has an hindex of 56, co-authored 293 publications receiving 13537 citations. Previous affiliations of Mark S. Pearce include University of Newcastle & National Institute for Health Research.

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Reliability and validity of ultrasound imaging of features of knee osteoarthritis in the community

TL;DR: Substantial to excellent agreement was found between ultrasound observers for the presence of osteophytes and measurement of effusion size; it was moderate to substantial for femoral cartilage thickness.
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Thyroid cancer in children and young adults in the North of England. Is increasing incidence related to the Chernobyl accident

TL;DR: Changes in incidence are consistent with a causal association with the Chernobyl accident although a greater effect in the younger rather than the older age group would have been anticipated, however, factors including improvements in ascertainment and earlier detection of tumours may also have contributed to the increasing incidence.
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Do Circumstances in Early Life Contribute to Tooth Retention in Middle Age

TL;DR: Promotion of a healthier adult lifestyle and continued improvements in oral hygiene would appear to be the public health interventions most likely to increase tooth retention in middle age.
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Study protocol: longitudinal study of the transition of young people with complex health needs from child to adult health services

TL;DR: This study will evaluate the effect of service components of transitional care, rather than evaluation of specific models that may be unsustainable or not generalisable, for young people with complex health needs making their transition.
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Postnatal Growth and DNA Methylation Are Associated With Differential Gene Expression of the TACSTD2 Gene and Childhood Fat Mass

TL;DR: The lack of association between fat mass and a methylation proxy SNP suggests that reverse causation or confounding may explain the initial association betweenFat mass and gene regulation and noncausal methylation patterns may still be useful predictors of later adiposity.