M
Marta Montpeyó
Researcher at Autonomous University of Barcelona
Publications - 4
Citations - 159
Marta Montpeyó is an academic researcher from Autonomous University of Barcelona. The author has contributed to research in topics: Autophagy & Mitophagy. The author has an hindex of 3, co-authored 3 publications receiving 59 citations.
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Journal ArticleDOI
Mutant HTT (huntingtin) impairs mitophagy in a cellular model of Huntington disease.
Sandra Franco-Iborra,Ainhoa Plaza-Zabala,Marta Montpeyó,David Sebastián,Miquel Vila,Miquel Vila,Marta Martinez-Vicente +6 more
TL;DR: The role of HTT during mitophagy is investigated and the impact of the expansion of its polyglutamine (polyQ) tract is analyzed, leading to the accumulation of damaged mitochondria and an increase in oxidative stress in Huntington disease.
Journal ArticleDOI
The Emerging Role of the Lysosome in Parkinson's Disease.
TL;DR: Evidence includes the presence of α-synuclein inclusions in the brain regions of patients with LSD and the identification of several lysosomal genes involved in LSD as genetic risk factors to develop PD.
Journal ArticleDOI
Impaired proteasome activity and neurodegeneration with brain iron accumulation in FBXO7 defect.
Marta Correa-Vela,Vincenzo Lupo,Marta Montpeyó,Paula Sancho,Anna Marcé-Grau,Jorge Hernández‐Vara,Alejandra Darling,Alison Jenkins,Sandra Fernández‐Rodríguez,Cristina Tello,Laura Ramírez-Jiménez,Belén Pérez,Angel Sanchez-Montanez,Alfons Macaya,María Jesús Sobrido,Marta Martinez-Vicente,Belén Pérez-Dueñas,Carmen Espinós +17 more
TL;DR: This novel FBXO7 phenotype associated with impaired proteasome activity overlaps with neurodegeneration with brain iron accumulation disorders.
Journal ArticleDOI
Lysosomal lipid alterations caused by glucocerebrosidase deficiency promote lysosomal dysfunction, chaperone-mediated-autophagy deficiency, and alpha-synuclein pathology
Alba Navarro-Romero,Irene Fernández-González,Jordi Riera,Marta Montpeyó,Mercé Albert-Bayo,Tresa López-Royo,Pablo Castillo-Sanchez,Clara Carnicer-Cáceres,José Antonio Arranz Amo,Laura Castillo-Ribelles,Eddie Pradas,Josefina Casas,Miquel Vila,Marta Martinez-Vicente +13 more
TL;DR: In this paper , a deep analysis of the consequences triggered by the presence of mutant GBA protein and the loss of β-glucocerebrosidase activity in different cellular compartments, focusing primarily on the lysosomal compartment, was performed.