M
Martin Genet
Researcher at École Polytechnique
Publications - 83
Citations - 1416
Martin Genet is an academic researcher from École Polytechnique. The author has contributed to research in topics: Finite element method & Medicine. The author has an hindex of 23, co-authored 73 publications receiving 1109 citations. Previous affiliations of Martin Genet include Université Paris-Saclay & French Institute for Research in Computer Science and Automation.
Papers
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Journal ArticleDOI
Distribution of normal human left ventricular myofiber stress at end diastole and end systole: a target for in silico design of heart failure treatments.
Martin Genet,Lik Chuan Lee,Rebecca Nguyen,Henrik Haraldsson,Gabriel Acevedo-Bolton,Zhihong Zhang,Liang Ge,Karen G. Ordovas,Sebastian Kozerke,Julius M. Guccione +9 more
TL;DR: This paper constructed personalized computational models of the left ventricles of five normal human subjects using magnetic resonance images and the finite-element method to construct reference maps of normal ventricular wall stress in humans that could serve as a target for in silico optimization studies of existing and potential new treatments for heart failure.
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On the structural, mechanical, and biodegradation properties of HA/β‐TCP robocast scaffolds
TL;DR: The findings from this work indicate that composite calcium phosphate scaffolds with customer-designed chemistry and architecture may be fabricated by a robotic-assisted deposition method.
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Second-order motion-compensated spin echo diffusion tensor imaging of the human heart: Motion-Compensated Cardiac DTI
Christian T. Stoeck,Constantin von Deuster,Constantin von Deuster,Martin Genet,David Atkinson,Sebastian Kozerke,Sebastian Kozerke +6 more
TL;DR: This study compared first‐order motion‐compensated diffusion‐weighted imaging during systolic contraction of the heart with the use of second‐order Motion-Compensated Diffusion encoding for in vivo cardiac microstructure probe.
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Modeling Pathologies of Diastolic and Systolic Heart Failure
TL;DR: The model naturally connects molecular events of parallel and serial sarcomere deposition with cellular phenomena of myofibrillogenesis and sarcomerogenesis to whole organ function and can predict characteristic secondary effects including papillary muscle dislocation, annular dilation, regurgitant flow, and outflow obstruction.
Journal ArticleDOI
Heterogeneous growth-induced prestrain in the heart.
Martin Genet,Martin Genet,Manuel K. Rausch,Lik Chuan Lee,Lik Chuan Lee,S Choy,Xuefeng Zhao,Ghassan S. Kassab,Ghassan S. Kassab,Sebastian Kozerke,Julius M. Guccione,Ellen Kuhl +11 more
TL;DR: It is demonstrated that including the effects of prestrain reduces the left ventricular stiffness by up to 40%, thus facilitating the ventricular filling, which has a significant impact on cardiac function.