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Martin J. J. Ronis

Researcher at LSU Health Sciences Center New Orleans

Publications -  200
Citations -  8492

Martin J. J. Ronis is an academic researcher from LSU Health Sciences Center New Orleans. The author has contributed to research in topics: Soy protein & Osteoblast. The author has an hindex of 53, co-authored 196 publications receiving 7769 citations. Previous affiliations of Martin J. J. Ronis include University of Arkansas for Medical Sciences & Louisiana State University.

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Soy Protein Isolate Reduces Hepatosteatosis in Yellow Avy/a Mice Without Altering Coat Color Phenotype

TL;DR: It is concluded that the effects of purified GEN differ from those of SPI when GEN equivalents are closely matched and SPI does not epigenetically regulate the agouti locus to shift the coat color phenotype in the same fashion as GEN alone.
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Limited therapeutic effect of N-acetylcysteine on hepatic insulin resistance in an experimental model of alcohol-induced steatohepatitis.

TL;DR: Antioxidant treatments reduce the severity of chronic alcohol-related steatohepatitis, possibly because of the decreased expression of inflammatory mediators and ceramide accumulation, but they do not restore insulin/IGF-1 signaling in liver, most likely due to persistent elevation of GM3 synthase expression.
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Effects of diet and ethanol on the expression and localization of cytochromes P450 2E1 and P450 2C7 in the colon of male rats.

TL;DR: It is indicated that CYP2E1 is present in colonic epithelial cells, and expression of colonic and hepatic microsomal CYP 2E1 and CyP2C7 was increased by chronic ethanol intake, which may provide a partial explanation for the mechanism underlying effects of diet and ethanol on colon cancer.
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Effects of short-term ethanol and nutrition on the hepatic microsomal monooxygenase system in a model utilizing total enteral nutrition in the rat.

TL;DR: Whether ethanol has demonstrable effects in the presence of dietary sources that promote normal growth rates is determined and two clinically relevant diets were selected.
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Inhibition of fetal bone development through epigenetic down-regulation of HoxA10 in obese rats fed high-fat diet

TL;DR: The results suggest that maternal obesity impairs fetal skeletal development through down‐regulation of the HoxA10 gene, which may lead to an increase in the prevalence of low bone mass in the offspring later in life.