M
Martin R. Lindley
Researcher at Loughborough University
Publications - 86
Citations - 3454
Martin R. Lindley is an academic researcher from Loughborough University. The author has contributed to research in topics: Bronchoconstriction & Eicosapentaenoic acid. The author has an hindex of 20, co-authored 83 publications receiving 2907 citations. Previous affiliations of Martin R. Lindley include Indiana University & Cardiff Metropolitan University.
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Journal ArticleDOI
The anti-inflammatory effects of exercise: mechanisms and implications for the prevention and treatment of disease
Michael Gleeson,Nicolette C. Bishop,David J. Stensel,Martin R. Lindley,Sarabjit S. Mastana,Myra A. Nimmo +5 more
TL;DR: The known mechanisms by which exercise — both acute and chronic — exerts its anti-inflammatory effects are focused on, and the implications of these effects for the prevention and treatment of disease are discussed.
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Fish oil supplementation reduces severity of exercise-induced bronchoconstriction in elite athletes.
TL;DR: It is suggested that dietary fish oil supplementation has a markedly protective effect in suppressing EIB in elite athletes, and this may be attributed to their antiinflammatory properties.
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Protective effect of fish oil supplementation on exercise-induced bronchoconstriction in asthma.
TL;DR: The data suggest that fish oil supplementation may represent a potentially beneficial nonpharmacologic intervention for asthmatic subjects with EIB, and was significantly reduced before and following exercise on the fish oil diet compared to the normal and placebo diets.
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Protective Effect of Fish Oil Supplementation on Exercise-Induced Bronchoconstriction in Asthmatic Subjects: 2160
TL;DR: The data suggest that fish oil supplementation may represent a potentially beneficial nonpharmacologic intervention for asthmatic subjects with EIB, and improved pulmonary function to below the diagnostic EIB threshold.
Journal Article
Dietary salt, airway inflammation, and diffusion capacity in exercise-induced asthma.
TL;DR: It is indicated that dietary salt loading enhances airway inflammation following exercise in asthmatic subjects, and that small salt-dependent changes in vascular volume and microvascular pressure might have substantial effects on airway function following Exercise in the face of mediator-induced increased vascular permeability.