M
Mary E. Wlodek
Researcher at University of Melbourne
Publications - 202
Citations - 4893
Mary E. Wlodek is an academic researcher from University of Melbourne. The author has contributed to research in topics: Offspring & Pregnancy. The author has an hindex of 35, co-authored 189 publications receiving 4251 citations. Previous affiliations of Mary E. Wlodek include University of Western Australia & St. Vincent's Institute of Medical Research.
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Journal ArticleDOI
Possible role of uterine contractions in the short-term fluctuations of plasma acth concentration in fetal sheep
TL;DR: It is speculated that increases in uterine activity and/or transient decreases in fetal PaO2 may contribute to short-term fluctuations in plasma ACTH in fetal sheep.
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Uteroplacental insufficiency leads to hypertension, but not glucose intolerance or impaired skeletal muscle mitochondrial biogenesis, in 12-month-old rats.
Melanie Tran,Margaret E. Young,Andrew J. Jefferies,Deanne H. Hryciw,Michelle M. Ward,Erica L. Fletcher,Mary E. Wlodek,Glenn D. Wadley,Glenn D. Wadley +8 more
TL;DR: Sex strongly impacts the cardiometabolic effects of growth restriction in 12‐month‐old rats and it is females who are at particular risk of developing long‐term hypertension following growth restriction.
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Maternal obesity in females born small: Pregnancy complications and offspring disease risk
Dayana Mahizir,Jessica F. Briffa,Deanne H. Hryciw,Glenn D. Wadley,Karen M. Moritz,Mary E. Wlodek +5 more
TL;DR: Early pregnancy interventions targeted to obese mothers may prevent their children from developing cardiometabolic disease in adulthood, including type 2 diabetes, obesity, and hypertension during pregnancy.
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Bladder contractions and micturition in fetal sheep: their relation to behavioral states
TL;DR: Monitoring in unanesthetized fetal sheep in utero during the last third of gestation shows that most spontaneous voids in the fetus begin during LV ECoG, suggesting that voiding is regulated by descending information from the brain.
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Preterm fetal growth restriction is associated with increased parathyroid hormone-related protein expression in the fetal membranes.
Narelle E Curtis,Roger G King,Jane M. Moseley,Patricia W. M. Ho,Gregory E. Rice,Mary E. Wlodek +5 more
TL;DR: Either parathyroid hormone-related protein messenger ribonucleic acid or protein expression, or both, was increased in the fetal membranes in association with fetal growth restriction in preterm but not term gestations, suggesting that parathyro hormone- related protein may be involved in the pathogenesis of preterm fetal growth restrictions.