M
Mary E. Wlodek
Researcher at University of Melbourne
Publications - 202
Citations - 4893
Mary E. Wlodek is an academic researcher from University of Melbourne. The author has contributed to research in topics: Offspring & Pregnancy. The author has an hindex of 35, co-authored 189 publications receiving 4251 citations. Previous affiliations of Mary E. Wlodek include University of Western Australia & St. Vincent's Institute of Medical Research.
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Journal ArticleDOI
Maternal growth restriction and stress exposure in rats differentially alters expression of components of the placental glucocorticoid barrier and nutrient transporters.
Jessica F. Briffa,Sogand S. Hosseini,Melanie Tran,Karen M. Moritz,James S. M. Cuffe,Mary E. Wlodek +5 more
TL;DR: The importance of informing pregnant women on effective ways to cope with stress during pregnancy to prevent adverse long-term disease outcomes in their children is highlighted.
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PTH/PTHrP receptor and mid-molecule PTHrP regulation of intrauterine PTHrP: PTH/PTHrP receptor antagonism increases SHR fetal weight.
Mary E. Wlodek,R. Di Nicolantonio,Kerryn T. Westcott,W. Farrugia,Patricia W. M. Ho,Jane M. Moseley +5 more
TL;DR: It is highlighted that an increase in endogenous uterine, placental and fetal plasma PTHrP following PTH/PTHRP receptor antagonism was associated with increased SHR fetal growth presumably by improving placental growth and function.
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Adrenal, metabolic and cardio‐renal dysfunction develops after pregnancy in rats born small or stressed by physiological measurements during pregnancy
Jean N. Cheong,James S. M. Cuffe,James S. M. Cuffe,Andrew J. Jefferies,Karen M. Moritz,Mary E. Wlodek +5 more
TL;DR: Investigating the effects of stress during pregnancy on the long‐term adrenal, metabolic and cardio‐renal health of female rats that were born small suggests that being born small or being stressed during pregnancy does not exacerbate the long-term adverse health outcomes after pregnancy.
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Fetal versus maternal determinants of the reduced fetal and placental growth in spontaneously hypertensive rats.
TL;DR: The SHR fetus exhibited reduced growth rate and placental size irrespective of maternal surrogate strain, suggesting that these measures are likely to be determined by the fetus or the placenta and, presumably, are independent of maternal blood pressure or altered electrolyte and hormonal milieu.
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Exercise improves metabolic function and alters the microbiome in rats with gestational diabetes
Dayana Mahizir,Jessica F. Briffa,Jennifer L. Wood,Kristina Anevska,Elisa L. Hill-Yardin,Elisa L. Hill-Yardin,Andrew J. Jefferies,Sogand Gravina,Gisella Mazzarino,Ashley E. Franks,Karen M. Moritz,Glenn D. Wadley,Mary E. Wlodek +12 more
TL;DR: It is suggested that maternal exercise and diet influence metabolic and microbiome dysfunction in females with GDM, which may impact long‐term maternal and offspring health.