scispace - formally typeset
M

Maryline Magnin-Robert

Researcher at University of the Littoral Opal Coast

Publications -  40
Citations -  1169

Maryline Magnin-Robert is an academic researcher from University of the Littoral Opal Coast. The author has contributed to research in topics: Medicine & Plant defense against herbivory. The author has an hindex of 16, co-authored 33 publications receiving 864 citations. Previous affiliations of Maryline Magnin-Robert include Centre national de la recherche scientifique & University of Reims Champagne-Ardenne.

Papers
More filters
Journal ArticleDOI

Grapevine trunk diseases: complex and still poorly understood

TL;DR: An overview of eutypa dieback, esca and botryosphaeria die back, the predominant grapevine trunk diseases worldwide, and their symptomatologies, characteristics of the different fungal species associated with them; and host‐pathogen interactions are presented.
Journal ArticleDOI

Biological control of Botrytis cinerea by selected grapevine-associated bacteria and stimulation of chitinase and β-1,3 glucanase activities under field conditions

TL;DR: Results have demonstrated an induced protection of grapevine against B. cinerea by selected bacteria under field conditions, and suggest that induced resistance could be related to a stimulation of plant defense reactions in a successive manner.
Journal ArticleDOI

Leaf stripe form of esca induces alteration of photosynthesis and defence reactions in presymptomatic leaves

TL;DR: The results suggest that grapevines may react precociously by reducing photosynthesis and triggering defence mechanisms in response to the leaf stripe form of esca.
Journal ArticleDOI

Modifications of Sphingolipid Content Affect Tolerance to Hemibiotrophic and Necrotrophic Pathogens by Modulating Plant Defense Responses in Arabidopsis

TL;DR: It is demonstrated that sphingolipids play an important role in plant defense, especially toward necrotrophic pathogens, and highlight a novel connection between the jasmonate signaling pathway, cell death, and sphingOLipids.