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Masatoshi Amako

Researcher at National Defense Medical College

Publications -  34
Citations -  791

Masatoshi Amako is an academic researcher from National Defense Medical College. The author has contributed to research in topics: Sciatic nerve & Distraction osteogenesis. The author has an hindex of 14, co-authored 33 publications receiving 736 citations.

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Effect of Static Stretching on Prevention of Injuries for Military Recruits

TL;DR: In this article, a prospective study was designed to evaluate whether static stretching can prevent training-related injuries in Japan Ground Self-Defense Force military recruits, where a total of 901 recruits between 1996 and 1998 were divided into two groups.
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Expression of bone morphogenetic proteins during mandibular distraction osteogenesis.

TL;DR: Data indicate that BMPs participate in the translation of mechanical stimuli into a biological response during mandibular distraction osteogenesis, a form of in vivo tissue engineering in which the gradual separation of cut bone edges results in the generation of new bone.
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Effects of osteogenic protein-1 on distraction osteogenesis in rabbits.

TL;DR: It appears that the lack of receptor protein in the target tissue impairs the effect of OP-1 given at the end of the distraction period, and OP- 1 could be more useful when applied early in the distraction phase.
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Comparison between partial and minimal medial epicondylectomy combined with decompression for the treatment of cubital tunnel syndrome

TL;DR: In this article, the authors compared surgical outcomes between partial medial epicondylectomy (14 patients) and minimal medial epidermal deprogramming (18 patients) combined with ulnar nerve decompression for the treatment of cubital tunnel syndrome.
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An fMRI study of musicians with focal dystonia during tapping tasks

TL;DR: Functional magnetic resonance imaging is performed to investigate the motor-related brain activity associated with musician’s dystonia and suggests that irregular neural activity patterns in dystonic subjects reflect dySTONic neural malfunction and consequent compensatory activity to maintain appropriate voluntary movements.