M
Masayuki Murata
Researcher at Kyushu University
Publications - 1211
Citations - 15753
Masayuki Murata is an academic researcher from Kyushu University. The author has contributed to research in topics: Network packet & Wireless sensor network. The author has an hindex of 51, co-authored 1163 publications receiving 14719 citations. Previous affiliations of Masayuki Murata include Tokyo Metropolitan University & Tokyo Institute of Technology.
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Journal ArticleDOI
Vip21/caveolin is a cholesterol-binding protein
Masayuki Murata,Johan Peränen,Rupert Schreiner,Felix T. Wieland,Teymuras V. Kurzchalia,Kai Simons +5 more
TL;DR: Findings suggest that VIP21/caveolin, through its cholesterol-binding properties, serves a specific function in microdomain formation during membrane trafficking in caveolae and apical transport vesicles.
Indoor Localization System using RSSI Measurement of Wireless Sensor Network based on ZigBee Standard.
TL;DR: To verify the validity of the previously reported autonomous indoor localization system in an actual environment, it was implemented in a wireless sensor network based on the ZigBee standard and showed that when the deployment density of sensor nodes was set to 0.27 nodes/ , the position estimation error was reduced.
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Crystal structure analyses of reduced (CuI) poplar plastocyanin at six pH values.
TL;DR: The structure of poplar plastocyanin in the reduced (CuI) state has been determined and refined, using counter data recorded from crystals at pH 3.8 and 7.8, and the trigonal geometry of the Cu atom strongly favours CuI, so that this form of the protein should be redox-inactive.
Performance of Alternate Routing Methods in All-Optical Switching Networks
TL;DR: In this article, the authors considered an alternate routing method with limited trunk reservation in which connections with more hops are prepared more alternate routes, and they showed that their method keeps good performance when compared with the existing alternate routing methods.
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Expression of the peroxisome proliferator activated receptor γ gene is repressed by DNA methylation in visceral adipose tissue of mouse models of diabetes
TL;DR: It is proposed that reduced expression of PPARγ owing to DNA methylation in adipocytes of the VAT may contribute to the pathogenesis of metabolic syndrome.