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Massimo Riccio

Researcher at University of Modena and Reggio Emilia

Publications -  70
Citations -  3492

Massimo Riccio is an academic researcher from University of Modena and Reggio Emilia. The author has contributed to research in topics: Stem cell & Cellular differentiation. The author has an hindex of 37, co-authored 70 publications receiving 3247 citations. Previous affiliations of Massimo Riccio include National Service of Learning & University of Bologna.

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Induction of long-term potentiation and depression is reflected by corresponding changes in secretion of endogenous brain-derived neurotrophic factor

TL;DR: This data indicates that suppression of BDNF by TrkB-IgG receptor bodies prevented LTP and low-frequency stimulations leading to LTD were accompanied by reductions in BDNF secretion that never lasted beyond the duration of the stimulation.
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Chlorpromazine equivalents: a consensus of opinion for both clinical and research applications

TL;DR: A consensus of available information is used to produce a working table of conversion for the most commonly used drugs to chlorpromazine equivalents and a user-friendly computer program has been developed from this information.
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Human dental pulp stem cells (hDPSCs): isolation, enrichment and comparative differentiation of two sub-populations.

TL;DR: This study characterized and compared two subpopulations of adult stem cells derived from human dental pulp, and suggested that STRO-1+/c-Kit+/CD34− hDPSCs and STROs might represent two distinct stem cell populations, with different properties.
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Ataxin-3 is Transported Into the Nucleus and Associates with the Nuclear Matrix

TL;DR: The results taken together with the finding of a nuclear localization signal in ataxin-3 indicate that the ataxIn-3 protein per se translocates to the nucleus and that an expanded glutamine repeat is not essential for this transport.
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Hippocampal neurons recycle BDNF for activity-dependent secretion and LTP maintenance

TL;DR: Evidence is provided that hippocampal neurons additionally recycle BDNF for activity‐dependent secretion, which means that endocytosed BDNF is fed back to the activity‐ dependent releasable pool required for LTP maintenance.