M
Mei-Li Wu
Researcher at National Pingtung University of Science and Technology
Publications - 14
Citations - 298
Mei-Li Wu is an academic researcher from National Pingtung University of Science and Technology. The author has contributed to research in topics: Methylglyoxal & Antigen. The author has an hindex of 8, co-authored 13 publications receiving 256 citations.
Papers
More filters
Journal ArticleDOI
Biosynthetic Mechanism of 2-Acetyl-1-pyrroline and Its Relationship with Δ1-Pyrroline-5-carboxylic Acid and Methylglyoxal in Aromatic Rice (Oryza sativa L.) Callus
TL;DR: Upregulation of P5CS in aromatic rice Tainung 72 may contribute to the increase of Delta(1)-pyrroline-5-carboxylic acid level and thus leads to the accumulation of an extra amount of 2-acetyl-1-pyr roline.
Journal ArticleDOI
Cinnamaldehyde enhances Nrf2 nuclear translocation to upregulate phase II detoxifying enzyme expression in HepG2 cells.
TL;DR: It is suggested that ERK1/2, Akt, and JNK pathways activated by cinnamaldehyde collectively control Nrf2 nuclear translocation and transcriptional activity, leading to the increase of phase II enzyme expression.
Journal ArticleDOI
δ1-Pyrroline-5-carboxylic Acid Formed by Proline Dehydrogenase from the Bacillus subtilis ssp. natto Expressed in Escherichia coli as a Precursor for 2-Acetyl-1-pyrroline
TL;DR: The data showed that P5C, derived from the conversion of l-proline by the purified recombinant PRODH, might react directly with methylglyoxal to form 2-AP, which represents the first report of a biological mechanism by which 2- AP may be synthesized in vitro byPRODH.
Journal ArticleDOI
Adjuvant effects of chicken interleukin-18 in avian Newcastle disease vaccine.
TL;DR: Full length and mature chicken IL-18 expressed using a prokaryotic system and using a eukaryoticSystem showed equivalent in vitro and in vivo biological activities, and all forms effectively enhanced cell-mediated and humoral immunity, suggesting possible future use as a potential adjuvant in chicken NDTC vaccine production.
Journal ArticleDOI
Induction of apoptosis by vitamin D2, ergocalciferol, via reactive oxygen species generation, glutathione depletion, and caspase activation in human leukemia Cells.
TL;DR: Findings suggest that ergocalciferol causes HL-60 apoptosis via a modulation of mitochondria involving ROS production, GSH depletion, caspase activation, and Fas induction, which may be feasible to develop chemopreventive agents from edible mushrooms or hop.