M
Melisa Moore
Researcher at Children's Hospital of Philadelphia
Publications - 37
Citations - 1914
Melisa Moore is an academic researcher from Children's Hospital of Philadelphia. The author has contributed to research in topics: Sleep disorder & Obstructive sleep apnea. The author has an hindex of 18, co-authored 33 publications receiving 1689 citations. Previous affiliations of Melisa Moore include University of Pennsylvania & Case Western Reserve University.
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Journal ArticleDOI
Mortality Associated with Short Sleep Duration: The Evidence, The Possible Mechanisms, and The Future
TL;DR: A social-ecological framework is put forth to explore five possible pathways for the relationship between sleep duration and mortality, and a four-point agenda for future research is concluded.
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The sleepy adolescent: causes and consequences of sleepiness in teens
Melisa Moore,Lisa J. Meltzer +1 more
TL;DR: The majority of adolescents do not obtain the recommended amount of sleep, resulting in significant daytime sleepiness, which underscores the importance of evaluating sleepy adolescents and treating any underlying sleep disorders.
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Correlates of adolescent sleep time and variability in sleep time: The role of individual and health related characteristics
TL;DR: Differences in sleep patterns in population sub-groups of adolescents may be important in understanding pediatric health risk profiles and boys, overweight, and minority adolescents may particularly benefit from interventions aimed at improving sleep patterns.
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A Meta-Analysis of the Effects of Psychological Interventions in Pediatric Oncology on Outcomes of Psychological Distress and Adjustment
TL;DR: Psychological interventions in pediatric oncology show promise in decreasing distress and improving the adjustment of parents of children with cancer but may have minimal effects for child outcomes.
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Sleep Disruptions in Parents of Children and Adolescents with Chronic Illnesses: Prevalence, Causes, and Consequences
Lisa J. Meltzer,Melisa Moore +1 more
TL;DR: Parents of youth with chronic illnesses experience sleep disruptions, providing a potential mechanism to explain elevated rates of negative daytime functioning found in previous studies.