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Miaoyun Qiu

Researcher at Guangzhou Medical University

Publications -  11
Citations -  344

Miaoyun Qiu is an academic researcher from Guangzhou Medical University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 4, co-authored 5 publications receiving 95 citations.

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Circular RNA circSATB2 promotes progression of non-small cell lung cancer cells

TL;DR: circSATB2 participated in the progression of NSCLC and was differentially expressed in lung cancer tissue and serumal exosomes, and may be potential biomarker for the diagnosis ofNSCLC.
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Circular RNA circBbs9 promotes PM2.5-induced lung inflammation in mice via NLRP3 inflammasome activation.

TL;DR: Results revealed that PM2.5 induced pulmonary inflammation through NLRP3 inflammasome activation regulated by the circBbs9-miR-30e-5p-Adar pathway, which provides a new target, circBBS9, for the assessment of lung inflammation and COPD exacerbation induced by PM 2.5 exposure.
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Circular RNA 406961 interacts with ILF2 to regulate PM2.5-induced inflammatory responses in human bronchial epithelial cells via activation of STAT3/JNK pathways.

TL;DR: RNA pull-down and mass spectrometry showed that circ_406961 inhibited activation of STAT3/JNK pathways via interacting with ILF2 protein, thereby inhibiting the PM2.5-induced BEAS-2B cell inflammation.
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CircRNA104250 and lncRNAuc001.dgp.1 promote the PM2.5-induced inflammatory response by co-targeting miR-3607-5p in BEAS-2B cells.

TL;DR: An underlying mechanism of airway inflammation by PM2.5 involving regulation of ncRNAs for the first time is uncovered, which provides further insights into the toxicological effects of PM 2.5.
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Gasdermin D-dependent platelet pyroptosis exacerbates NET formation and inflammation in severe sepsis

TL;DR: Su et al. as discussed by the authors showed that platelet-specific GSDMD deficiency in platelets or pharmacological inhibition of S100A8/A9 using Paquinimod can break this detrimental feedback loop, thus ameliorating excessive NET-mediated inflammation in mouse models of severe sepsis.