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Michael D. Prados

Researcher at University of California, San Francisco

Publications -  466
Citations -  57545

Michael D. Prados is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Glioma & Temozolomide. The author has an hindex of 107, co-authored 444 publications receiving 51418 citations. Previous affiliations of Michael D. Prados include Harvard University & University of Texas at Austin.

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Effect of disease burden on health-related quality of life in patients with malignant gliomas.

TL;DR: Most of the symptoms were likely due to recurrence, but previous radiation therapy and on-going corticosteroid treatment may have also been casual factors for fatigue, whereas uncertainty about the future and pain were probably nonspecific for brain cancer.
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Management of chiasmal and hypothalamic gliomas of infancy and childhood with chemotherapy.

TL;DR: Preliminary results suggest that nitrosourea-based cytotoxic regimens are useful for the initial treatment of children with chiasmal/hypothalamic gliomas, and allow potentially harmful radiation therapy to be deferred until progression of disease.
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Individual patient-specific immunity against high-grade glioma after vaccination with autologous tumor derived peptides bound to the 96 KD chaperone protein.

TL;DR: These data provide the first evidence in humans of individual patient-specific immune responses against autologous tumor derived peptides bound to HSP-96, which is used to safely immunize patients with recurrent GBM.
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Radiation response and survival time in patients with glioblastoma multiforme

TL;DR: The authors studied a cohort of 301 patients who were initially treated according to uniform clinical protocols and found that neuroimaging obtained within 3 days after surgery served as a better baseline for assessment of radiation response than images obtained later.
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18-fluorodeoxyglucose uptake and survival of patients with suspected recurrent malignant glioma

TL;DR: After intensive initial radiation therapy for malignant glioma, magnetic resonance imaging (MRI) and computerized tomography (CT) cannot distinguish tumor progression from radiation injury.