M
Michael D. Prados
Researcher at University of California, San Francisco
Publications - 466
Citations - 57545
Michael D. Prados is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Glioma & Temozolomide. The author has an hindex of 107, co-authored 444 publications receiving 51418 citations. Previous affiliations of Michael D. Prados include Harvard University & University of Texas at Austin.
Papers
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Journal ArticleDOI
Ed-36inherited variants near terc and tert are associated with longer telomeres and increased glioma risk: genome-wide association results from the ucsf adult glioma study and the engage consortium telomere group
Kyle M. Walsh,Veryan Codd,Ivan Smirnov,Terri Rice,Paul A. Decker,Helen M. Hansen,Annette M. Molinaro,Melike Pekmezci,Tarik Tihan,Mitchell S. Berger,Susan M. Chang,Michael D. Prados,Dan Lachance,Brian P. O'Neill,Pim van der Harst,John K. Wiencke,Nilesh J. Samani,Robert B. Jenkins,Margaret Wrensch +18 more
TL;DR: The identification of alleles that are strongly associated with both increased glioma risk and longer telomeres suggests that heritable variation in telomere length or telomerase activity may underlieglioma susceptibility.
Book ChapterDOI
P41 01 tp – the importance of thermal dose in the outcome of erain thermoradiotherapy
Patricia Sneed,Philip H. Gutin,Paul R. Stauffer,Theodore L. Phillips,Michael D. Prados,Keith A. Weaver,S. A. Suen,David K. Ahn,Kathleen R. Lamborn,David A. Larson,William M. Wara +10 more
Journal ArticleDOI
Geno-21brca1 protein expression predicts survival in glioblastoma patients from a nrg oncology/rtog cohort
Maria Vassilakopoulou,Maria Vassilakopoulou,Minhee Won,Walter J. Curran,Luis Souhami,Michael D. Prados,Corey J. Langer,David L. Rimm,Jason A. Hanna,Jason A. Hanna,Veronique Neumeister,William Smart,Aidnag Z. Diaz,James M Atkins,Lydia Komarnicky,Christopher J. Schultz,Steven P. Howard,Adam P. Dicker,Jonathan P.S. Knisely,Jonathan P.S. Knisely +19 more
TL;DR: BRCA1 protein expression was an important prognostic determinant in a cohort of glioblastoma patients treated with surgery followed by chemo-radio-therapy, and may imply that low BRCA 1 in the tumor, and the consequent low level of DNA repair, causes vulnerability of the cancer cells to treatment.
Journal ArticleDOI
EPCT-08. Disease-specific working groups within the Pacific Pediatric Neuro-Oncology Consortium (PNOC) and Children’s Brain Tumor Network (CBTN) facilitate multi-disciplinary collaboration and translation of innovative strategies in pediatric neuro-oncology
TL;DR: Multidisciplinary working groups within international consortia facilitate efficient translation of multi-pronged strategies to improve the care of children and young adults with central nervous system tumors.