Michael J. Baum

TL;DR: The results suggest that the termination of behavioral estrus is not associated with increased metabolic activity in central serotonergic neurons, and Hypothalamic and cortical concentrations of 5-HT and 5-HIAA were equivalent in the two groups.

TL;DR: It is hypothesized that prenatal exposure of the male ferret to estrogen, derived from the neural aromatization of circulating androgen, may sensitize the developing brain to the subsequent masculinizing action of testosterone shortly after birth.

TL;DR: Four experiments were performed to determine whether the activational effects of two behaviorally active neural metabolites of testosterone, estradiol (E2) and 5α-dihydrotestosterone (DHT), on male rats' sexual behavior possibly result from the action of either steroid at dopaminergic or serotoninergic synapses.

TL;DR: Evidence is reviewed that establishes an essential role for estradiol, synthesized locally via aromatization of testosterone in the adult as well as the developing male nervous system of rats, ferrets, and mice, in both the organization and adult activation of neural circuits that control appetitive aswell as consummatory components of masculine sexual behavior.

TL;DR: It is concluded that P probably disrupts sexual interaction in monkeys by antagonizing the facilitatory effect of E 2 on production of a vaginal factor which otherwise enhances sexual attractivity.