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Michael J. Baum

Researcher at Boston University

Publications -  370
Citations -  27670

Michael J. Baum is an academic researcher from Boston University. The author has contributed to research in topics: Breast cancer & Olfactory system. The author has an hindex of 68, co-authored 368 publications receiving 26574 citations. Previous affiliations of Michael J. Baum include University of Cambridge & Erasmus University Rotterdam.

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Copulation in Castrated Male Rats following Combined Treatment with Estradiol and Dihydrotestosterone

TL;DR: Castrated male rats injected daily with 2 micrograms of estradiol benzoate (EB) combined with 200 microgramS of dihydrotestosterone propionate (DHTP) displayed masculine mating behavior which was indistinguishable from that of other castrates treated with 200 milligrams of testosterone propionates.
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Retrospective Analysis of Time to Recurrence in the ATAC Trial According to Hormone Receptor Status: An Hypothesis-Generating Study

TL;DR: Time to recurrence was longer for anastrozole- than tamoxifen-treated patients in both ER+/PgR+ and ER-/Pg R- subgroups, but the benefit was substantially greater in the PgR- subgroup.
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Genetic and hormonal factors modulate spreading depression and transient hemiparesis in mouse models of familial hemiplegic migraine type 1

TL;DR: It is shown that transgenic mice expressing R192Q or S218L FHM1 mutations have increased SD frequency and propagation speed; enhanced corticostriatal propagation; and, similar to the human F HM1 phenotype, more severe and prolonged post-SD neurological deficits.
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A direct main olfactory bulb projection to the 'vomeronasal' amygdala in female mice selectively responds to volatile pheromones from males.

TL;DR: The presence of a direct MOB‐to‐medial amygdala pathway in mice and other mammals could enable volatile, opposite‐sex pheromones to gain privileged access to diencephalic structures that control mate recognition and reproduction.
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Neuroendocrine regulation of GnRH release in induced ovulators.

TL;DR: A complete understanding of the neuroendocrine control of ovulation will only be achieved by comparative studies of several animal model systems in which mating-induced as well as spontaneous, hormonally stimulated activation of GnRH neurons drives the preovulatory LH surge.