M
Michael J. Weyant
Researcher at University of Colorado Denver
Publications - 145
Citations - 3477
Michael J. Weyant is an academic researcher from University of Colorado Denver. The author has contributed to research in topics: Lung cancer & Paraplegia. The author has an hindex of 27, co-authored 137 publications receiving 2992 citations. Previous affiliations of Michael J. Weyant include Anschutz Medical Campus & Memorial Sloan Kettering Cancer Center.
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Journal ArticleDOI
Results of chest wall resection and reconstruction with and without rigid prosthesis.
Michael J. Weyant,Manjit S. Bains,Ennapadam Venkatraman,Robert J. Downey,Bernard J. Park,Raja M. Flores,Nabil P. Rizk,Valerie W. Rusch +7 more
TL;DR: The incidence of respiratory failure is lower than previously reported and may relate to the use of rigid repair for defects likely to cause a flail segment.
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Adjuvant Systemic Therapy and Adjuvant Radiation Therapy for Stage I to IIIA Completely Resected Non–Small-Cell Lung Cancers: American Society of Clinical Oncology/Cancer Care Ontario Clinical Practice Guideline Update
Mark G. Kris,Laurie E. Gaspar,Jamie E. Chaft,Erin B. Kennedy,Christopher G. Azzoli,Peter M. Ellis,Steven H. Lin,Harvey I. Pass,Rahul Seth,Frances A. Shepherd,David R. Spigel,John R. Strawn,Yee C. Ung,Michael J. Weyant +13 more
TL;DR: The guideline provides information on factors other than stage to consider when making a recommendation for adjuvant chemotherapy, including tumor size, histopathologic features, and genetic alterations, which are not recommended for patients with stage IA disease.
Journal ArticleDOI
Expression of functional Toll-like receptors 2 and 4 in human aortic valve interstitial cells: potential roles in aortic valve inflammation and stenosis.
Xianzhong Meng,Lihua Ao,Yong Song,Ashok N. Babu,Xiaoping Yang,Maorong Wang,Michael J. Weyant,Charles A. Dinarello,Joseph C. Cleveland,David A. Fullerton +9 more
TL;DR: It is demonstrated for the first time that HAVICs express TLR2 and TLR4 and that stimulation of HAVics by PGN or LPS induces the expression of proinflammatory mediators and the upregulation of osteogenesis-associated factors.
Journal Article
(+)-Catechin inhibits intestinal tumor formation and suppresses focal adhesion kinase activation in the min/+ mouse.
TL;DR: The natural abundance and favorable bioavailability of (+)-catechin make it a promising addition to the list of potential colorectal cancer chemopreventive agents.
Journal Article
Reciprocal Expression of ERα and ERβ Is Associated with Estrogen-mediated Modulation of Intestinal Tumorigenesis
Michael J. Weyant,Adelaide M. Carothers,Najjia N. Mahmoud,H. Leon Bradlow,Helen Remotti,Robyn T. Bilinski,Monica M. Bertagnolli +6 more
TL;DR: It is suggested that endogenous estrogens protect against Apc-associated tumor formation and that tumor prevention by 17beta-estradiol is associated with an increase in ERbeta and a decrease in ERalpha expression in the target tissue.