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Millie D. Long

Researcher at University of North Carolina at Chapel Hill

Publications -  253
Citations -  7105

Millie D. Long is an academic researcher from University of North Carolina at Chapel Hill. The author has contributed to research in topics: Inflammatory bowel disease & Medicine. The author has an hindex of 38, co-authored 180 publications receiving 4847 citations. Previous affiliations of Millie D. Long include Boston University.

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ACG Clinical Guideline: Ulcerative Colitis in Adults

TL;DR: These guidelines indicate the preferred approach to the management of adults with UC and represent the official practice recommendations of the American College of Gastroenterology.
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Risk of Melanoma and Nonmelanoma Skin Cancer Among Patients With Inflammatory Bowel Disease

TL;DR: Inflammatory bowel disease is associated with an increased incidence of melanoma and nonmelanoma skin cancer, and the risk of NMSC is increased by use of biologics and thiopurines, respectively, and patients with IBD should be counseled and monitored for skin cancer.
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ACG Clinical Guideline: Management of Irritable Bowel Syndrome.

TL;DR: This first-ever American College of Gastroenterology clinical guideline for the management of IBS recommends that a positive diagnostic strategy as compared to a diagnostic strategy of exclusion be used to improve time to initiating appropriate therapy and recommends gut-directed psychotherapy to treat global IBS symptoms.
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Increased risk for non-melanoma skin cancer in patients with inflammatory bowel disease.

TL;DR: Patients with inflammatory bowel disease, especially those who receive thiopurines, are at risk for non-melanoma skin cancer (NMSC), and Appropriate counseling and monitoring of such patients with IBD are recommended.
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A clinical review of recent findings in the epidemiology of inflammatory bowel disease

TL;DR: Current hypotheses as to the development of IBD are reviewed, as are a host of environmental cofactors that have been investigated as both protective and inciting factors for IBD onset and the role of these factors in disease course.