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Minoo Rassoulzadegan

Researcher at French Institute of Health and Medical Research

Publications -  125
Citations -  6564

Minoo Rassoulzadegan is an academic researcher from French Institute of Health and Medical Research. The author has contributed to research in topics: Gene & Epigenetics. The author has an hindex of 36, co-authored 117 publications receiving 6172 citations. Previous affiliations of Minoo Rassoulzadegan include University of Nice Sophia Antipolis & Erciyes University.

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RNA-mediated non-mendelian inheritance of an epigenetic change in the mouse

TL;DR: This work reports a similar modification of the mouse Kit gene in the progeny of heterozygotes with the null mutant Kittm1Alf (a lacZ insertion), identifying an unexpected mode of epigenetic inheritance associated with the zygotic transfer of RNA molecules.
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Peroxisome proliferator-activated receptor δ controls muscle development and oxydative capability

TL;DR: In this paper, the role of peroxisome proliferator-activated receptors (PPARs) in muscle development and adaptive response to environmental changes was investigated using a CRE-Lox recombination approach.
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Subnuclear localization of WT1 in splicing or transcription factor domains is regulated by alternative splicing

TL;DR: This work demonstrates colocalization of WT1 with splicing factors in the fetal kidney and testis and in expressing cell lines and proposes that WT1 plays roles in posttranscriptional processing of RNA as well as in transcription.
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The roles of individual polyoma virus early proteins in oncogenic transformation

TL;DR: The expression in normal rat cells of modified polyoma virus genomes, separately encoding large T, middle T or small T antigens, has allowed the investigation of the roles of these proteins in oncogenic transformation.
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RNA-mediated paternal heredity of diet-induced obesity and metabolic disorders

TL;DR: It is shown that microinjection of either testis or sperm RNA of male mice fed a Western-like diet into naive one-cell embryos leads to the establishment of the Western- like diet-induced metabolic phenotype in the resulting progenies, whereas RNAs prepared from healthy controls did not.