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Mohammad Nasiruddin Rana

Researcher at Zhejiang University

Publications -  12
Citations -  254

Mohammad Nasiruddin Rana is an academic researcher from Zhejiang University. The author has contributed to research in topics: Oxidative stress & Tumor necrosis factor alpha. The author has an hindex of 5, co-authored 10 publications receiving 149 citations. Previous affiliations of Mohammad Nasiruddin Rana include Walailak University & International Islamic University, Chittagong.

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Toxicodynamics of Lead, Cadmium, Mercury and Arsenic- induced kidney toxicity and treatment strategy: A mini review.

TL;DR: Oxidative Stress is the key mechanism involved in Lead, Mercury, Cadmium and Arsenic-induced kidney toxicity and possible effectiveness of plants and plants derived compound against heavy metals is due to their antioxidant activity.
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In vivo sedative activity of methanolic extract of Stericulia villosa Roxb. leaves.

TL;DR: It can be conclude that the methanolic extract of Sterculia villosa leaves posses significant a sedative property that may lead to new drug development and further investigation is necessary to understand the underlying mechanisms and to isolate the active principles.
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Xanthones protects lead-induced chronic kidney disease (CKD) via activating Nrf-2 and modulating NF-kB, MAPK pathway.

TL;DR: In silico prediction of activity study showed that protective role of xanthones could be due to its efficacy to activate the Nrf-2, regulate the intracellular [Ca2+], as well as downregulate the NF-kB, MAPK pathway.
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Tithonia diversifolia aqueous fraction plays a protective role against alloxan-induced diabetic mice via modulating GLUT2 expression

TL;DR: This paper evaluated the anti-diabetic effect of Tithonia diversifolia aqueous fraction (TdF) in alloxan-induced diabetic mice through an investigation of glucose transporter 2 (GLUT2) expression.
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Electroacupuncture ameliorates cardiopulmonary bypass induced apoptosis in lung via ROS/Nrf2/NLRP3 inflammasome pathway.

TL;DR: These findings indicate that EAc had a critical anti-apoptotic role by suppression of ROS/Nrf2/NLRP3 inflammasome pathway and might be a possible therapeutic treatment for CPB-induced acute lung injury.