scispace - formally typeset
M

Mu Qin

Researcher at Shanghai Jiao Tong University

Publications -  18
Citations -  3791

Mu Qin is an academic researcher from Shanghai Jiao Tong University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 4, co-authored 6 publications receiving 2708 citations.

Papers
More filters
Journal ArticleDOI

Association of Cardiac Injury With Mortality in Hospitalized Patients With COVID-19 in Wuhan, China.

TL;DR: The association between cardiac injury and mortality in patients with COVID-19 was analyzed and it was found that patients with cardiac injury had a higher proportion of multiple mottling and ground-glass opacity in radiographic findings than those without cardiac injury.
Journal ArticleDOI

Characteristics and clinical significance of myocardial injury in patients with severe coronavirus disease 2019.

TL;DR: The risk of in-hospital death among patients with severe COVID-19 can be predicted by markers of myocardial injury, and was significantly associated with senior age, inflammatory response, and cardiovascular comorbidities.
Journal ArticleDOI

Chronic N‐Methyl‐d‐Aspartate Receptor Activation Induces Cardiac Electrical Remodeling and Increases Susceptibility to Ventricular Arrhythmias

TL;DR: N‐Methyl‐d‐aspartate receptors, also known as NMDA Receptors or NMDAR, are glutamate receptors that control calcium ion channels and regulate synaptic plasticity.
Journal ArticleDOI

Coronavirus Disease 2019 (COVID-19) and Cardiac Injury-Reply.

TL;DR: The findings of Shi et al1 exploring cardiac injury in patients with confirmed coronavirus disease 2019 (COVID-19) indicated that prominent comorbidities in those with cardiac injury included hypertension, diabetes, coronary disease, heart failure, and cerebrovascular disease—all more prevalent in African American individuals.
Journal ArticleDOI

CaMKII in Regulation of Cell Death During Myocardial Reperfusion Injury.

TL;DR: In this paper, a brief overview of CaMKII with various posttranslational modifications and its properties in myocardial I/R injury is presented, focusing on the molecular mechanism of CAMKII involved in regulation of cell death induced by post-MI reperfusion including necroptosis and pyroptosis of cardiomyocyte.